<i>Amanita ibotengutake</i> Intoxication Treated With Plasma Exchange

Hiroshima, Yuko; Nakae, Hajime; Gommori, Kunio

doi:10.1111/j.1744-9987.2010.00862.x

Cited by 7 publications

(7 citation statements)

References 3 publications

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“…Ibotenic acid is a glutamate agonist, acting at N -methyl-D-aspartate (NMDA) receptors. Muscimol is a GABA A agonist [2,52]. Symptoms occur 30-180 min after ingestion.…”

Section: Inebriating Syndromesmentioning

confidence: 99%

“…Symptoms occur 30-180 min after ingestion. Symptoms usually peak at 2-3 h and disappear within 6-9 h [39,52]. Vomiting has been reported.…”

Section: Inebriating Syndromesmentioning

confidence: 99%

“…Alterations of time and space perception, auditory and visual hallucinations, and ataxia have been reported [51][52][53][54][55][56]. Rarely, tachycardia, hypertension, mydriasis, or flushing are seen [16,39].…”

Section: Inebriating Syndromesmentioning

confidence: 99%

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Mycetism: A Review of the Recent Literature

Graeme

2014

J. Med. Toxicol.

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Approximately 100 of the known species of mushrooms are poisonous to humans. New toxic mushroom species continue to be identified. Some species initially classified as edible are later reclassified as toxic. This results in a continually expanding list of toxic mushrooms. As new toxic species are identified, some classic teachings about mycetism no longer hold true. As more toxic mushrooms are identified and more toxic syndromes are reported, older classification systems fail to effectively accommodate mycetism. This review provides an update of myscetism and classifies mushroom poisonings by the primary organ system affected, permitting expansion, as new, toxic mushroom species are discovered.

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“…Ibotenic acid is a glutamate agonist, acting at N -methyl-D-aspartate (NMDA) receptors. Muscimol is a GABA A agonist [2,52]. Symptoms occur 30-180 min after ingestion.…”

Section: Inebriating Syndromesmentioning

confidence: 99%

“…Symptoms occur 30-180 min after ingestion. Symptoms usually peak at 2-3 h and disappear within 6-9 h [39,52]. Vomiting has been reported.…”

Section: Inebriating Syndromesmentioning

confidence: 99%

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Mycetism: A Review of the Recent Literature

Graeme

2014

J. Med. Toxicol.

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“…Finally, multiple dose activated charcoal administered until at least day 4 after ingestion removes the amatoxins from the enterohepatobiliary recirculation and thereby improves total toxin clearance [ 12 ]. Plasma exchange has also been discussed [ 13 , 14 ], but there is little data about the risk-benefit-ratio. The effect of all of these treatment modalities on late gastrointestinal complications however is, to our knowledge, unknown.…”

Section: Discussionmentioning

confidence: 99%

Ulcerating Ileocolitis in Severe Amatoxin Poisoning

Hilty

Halama

Zimmermann

et al. 2015

Case Reports in Gastrointestinal Medicine

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Amatoxin poisoning is still associated with a great potential for complications and a high mortality. While the occurrence of acute gastroenteritis within the first 24 hours after amatoxin ingestion is well described, only very few descriptions of late gastrointestinal complications of amatoxin poisoning exist worldwide. We present the case of a 57-year-old female patient with severe amatoxin poisoning causing fulminant but reversible hepatic failure that on day 8 after mushroom ingestion developed severe abdominal pain and watery diarrhea. Ulcerating ileocolitis was identified by computed tomography identifying a thickening of the bowel wall of the entire ileum and biopsies taken from the ileum and large bowel revealing distinct ileitis and proximally accentuated colitis. The absence of discernible alternative etiologies such as infectious agents makes a causal relationship between the ulcerating ileocolitis and the amatoxin poisoning likely. Diarrhea and varying abdominal pain persisted over several weeks and clinical follow-up after six months showed a completely symptom-free patient. The case presented highlights the importance to consider the possibility of rare complications of Amanita intoxication in order to be able to respond to them early and adequately.

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“…Ibotenic acid and muscimol are the poisons. Amanita muscaria, Amanita patherina, and Amanita ibotengutak are just a few of the mushrooms that are involved [14].…”

Section: Neurotoxic Mushroommentioning

confidence: 99%

Six groups of poisonous mushrooms: classified according to clinical symptoms

Yu¹

2022

HSET

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Since they have been consumed for a very long time, mushrooms have been linked to numerous ailments that are primarily toxin-induced. The precision and speed of diagnosis will be hampered by a lack of knowledge of the signs of mushroom poisoning, which poses a serious risk to public health and safety. This paper mainly discus cytotoxic, neurotoxic, myotoxic, metabolic, endocrine and related toxicity, gastrointestinal irritant, and miscellaneous adverse reactions of mushrooms, indicating that mushrooms can be categorized based on the kind of harmful reaction, which is helpful for the evaluation and analysis of clinical problems. By having a better understanding of various symptoms, one can make wiser decisions and develop effective coping mechanisms in the event of poisoning, thereby minimizing the damage caused by mushrooms and refocusing attention on the investigation of whether mushrooms may have any potential benefits.

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Amanita ibotengutake Intoxication Treated With Plasma Exchange

Cited by 7 publications

References 3 publications

Mycetism: A Review of the Recent Literature

Mycetism: A Review of the Recent Literature

Ulcerating Ileocolitis in Severe Amatoxin Poisoning

Six groups of poisonous mushrooms: classified according to clinical symptoms

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