I. Correlation of electrocardiographic and pathologic findings in anteroseptal infarction

Myers, Gordon B.; Klein, Howard A.; Stofer, Bert E.

doi:10.1016/0002-8703(48)90690-5

Cited by 160 publications

(30 citation statements)

References 29 publications

Supporting

Mentioning

Contrasting

Unclassified

Order By: Relevance

“…These findings are in keeping with patho logical studies that show that complete ob struction of the LAD artery causes total ne crosis and then fibrosis of the distal two thirds of the anterior wall, the apex and dis tal quarter of the inferior wall of the left ven tricle [9,10,19]. Angiographically, this is demonstrated as defective contraction, often with outward bulging of areas of necrotic or compliant fibrous tissue.…”

Section: Defective Spatial Contractionsupporting

confidence: 87%

Quantitation of Variations in Regional Left Ventricular Function in Isolated Disease of the Left Anterior Descending Coronary Artery

Sapoznikov

Welber²,

Lotan³

et al. 1987

Cardiology

View full text Add to dashboard Cite

The pattern of contraction of the endocardial wall of the left ventricle in the right anterior oblique cineangiogram was studied by using a frame by frame radial technique and a fixed centroid without correction for rotation and translation motion during the cardiac cycle. Spatial defects of contraction were quantitated by measuring the shortening fraction of each radius and temporospatial defects by using a time-contraction integral. Twelve normal subjects were used as a basis for comparison. Thirty-two patients with isolated disease of the left anterior descending (LAD) coronary artery were divided into seven arbitrary clinicopathological subsets. Five subsets showed significant quantitative differences in contraction from the normal subjects but there was no significant difference between the subsets. They had a typical defect of contraction in the distal two thirds of the anterior wall, the apex and distal quarter of the inferior wall of the left ventricle. The subsets included: (1) patients who had undergone an anterior myocardial infarction and who had total occlusion of the LAD artery and a large anterior infarction on ECG; (2) patients with a previous classical myocardial infarction but with only 95 % residual narrowing of the LAD; (3) patients with an anterior infarction and total occlusion of the LAD with return of the R waves in the anterior precordial leads; (4) patients with anterior infarction, LAD obstruction and left bundle branch block and (5) patients with anterior infarction but with early successful reperfusion with intracoronary streptokinase. Two other subsets, (1) patients with total obstruction of the LAD without a clinical myocardial infarction or (2) subtotal occlusion of the LAD without infarction, had mild left ventricular dysfunction at rest and did not differ significantly from normal.

show abstract

Section: Defective Spatial Contractionsupporting

confidence: 87%

Quantitation of Variations in Regional Left Ventricular Function in Isolated Disease of the Left Anterior Descending Coronary Artery

Sapoznikov

Welber²,

Lotan³

et al. 1987

Cardiology

View full text Add to dashboard Cite

show abstract

“…However, despite the fact that the ECG is quite reliable in detecting AAMI [1][2][3][4][5][6] or infarc tion caused by LAD artery obstruction [7,8], the relation between ECG 'anteroseptal wall involvement' and the anatomic site of the infarction or LAD occlusion is not clear. Previous works dealing with the electrocardio graphic and anatomic [1][2][3][4][5][6] or angiographic [7,14] corre lations in patients with AAMI did not investigate the ECG in the acute phase of the infarction. Recently, Strauss and Green [14] using the QRS morphology, con cluded that the correlation is limited.…”

Section: Discussionmentioning

confidence: 99%

“…The electrocardiogram (ECG) is quite reliable in de tecting anterior wall myocardial infarction (AAMI) [1][2][3][4][5][6] or infarction caused by left anterior descending (LAD) artery obstruction [7,8]. Based on the ECG, several subtypes of AAMI are recognized (anteroseptal, anterolateral, apical, etc.)…”

Section: Introductionmentioning

confidence: 99%

Can We Differentiate by the Admission Electrocardiogram between Anterior Wall Acute Myocardial Infarction due to a Left Anterior Descending Artery Occlusion Proximal to the Origin of the First Septal Branch and a Postseptal Occlusion?

Birnbaum¹,

Herz²,

Solodky³

et al. 1994

Am J Noninvas Cardiol

View full text Add to dashboard Cite

The objective of this study was to assess the value of ST changes in the various electrocardiographic (ECG) leads in predicting the location of the left anterior descending (LAD) coronary artery obstruction relative to the origin of the first septal branch in patients with anterior wall acute myocardial infarction (AAMI). Patients admitted to the coronary care unit with AAMI who underwent coronary angiography within 31 days of hospitalization were evaluated. The admission 12-lead ECG was evaluated for ST segment amplitudes in every lead at 0.08 s after the J point. The coronary angiogram was evaluated for the site and severity of luminal narrowing of the coronary arteries. The site of the culprit lesion in the LAD artery was determined relative to the origin of the first septal branch. A total of 111 consecutive patients were included. In 4 patients no lesion was identified in the LAD artery. Of the remaining 107 patients, 38 had a LAD lesion proximal to the origin of the first septal branch. No statistically significant differences were observed in ST amplitude in the precordial and lateral leads between the two groups. The magnitude of ST depression in the inferior leads is higher in patients with a preseptal lesion than in patients with a distal lesion (–1.05 ± 0.87 vs. –0.20 ± 0.82 mm, –1.61 ± 1.28 vs. –0.44 ± 0.96 mm, and –1.47 ± 1.13 vs. –0.33 ± 0.83 mm, for leads II, III and aVF, respectively, p < 0.0001). An ST depression of more than 1 mm in leads II, III and aVF has a predictive value for a preseptal lesion of 62% (p < 0.000005), 67% (p < 0.000004), and 74% (p < 0.000001), respectively, while most of the patients without ST depression in the inferior leads have a distal LAD artery lesion (82, 86 and 87% in leads II, III and aVF, respectively). ST elevation in the anteroseptal leads (V1 and V2) in the admission ECG does not differentiate between patients with an LAD artery occlusion proximal and distal to the origin of the first septal branch. The finding of isoelectric ST or ST elevation in the inferior leads is suggestive of a distal occlusive lesion in the LAD artery.

show abstract

“…In some of these studies the infarction was demonstrated at autopsy (Horan et al 1971 ; Levine and Phillips, 195 I ;Myers et al, 1948aMyers et al, , b, 1949a 1972, 1973Young et al, 1975). These studies give conflicting results regarding the diagnostic value of the ECG as a predictor of myocardial infarction with sensitivities ranging from 36% (Arkin et al, 1979;Wolff et al, 1961) to 87% (Young rt al., 1975) for anterior infarction and from 35% (Arkin et al, 1979) to 63% (Howard et al, 1976) for inferior infarction.…”

Section: Introductionmentioning

confidence: 99%

Accuracy of various techniques in the measurement of the duration of the q wave: A possible source of error in diagnosing myocardial infarction by electrocardiography

Mazzoleni¹,

DeMaria²

1983

Clinical Cardiology

View full text Add to dashboard Cite

Summary:The method of measuring the width of the Q wave on electrocardiogram is one of the variables contributing to conflicting findings regarding the accuracy of the ECG in the diagnosis of myocardial infarction.This study assesses the accuracy of a variety of methods of measuring the width of the Q wave recorded by direct-writing electrocardiographic instruments. The assessment was made by comparing the width of the Q wave as inscribed by thermal direct-writing electrocardiographs to the width of the Q wave as measured from an oscilloscopic display, the latter representing the "true" width of the Q wave. The measurement of the width as obtained from the upper edge of the tracing obtained with direct-writing electrocardiographs underestimated the true width of the Q wave, while the opposite was the case by measuring the width along the lower edge of the tracing. The most reliable ways to obtain the true width of the Q wave are: (1) to average the measurements as obtained along the upper and lower edge, (2) to measure the width along an ideal line in the middle of the tracing, or (3) to measure the width along the trailing edge of the deflection.

show abstract

I. Correlation of electrocardiographic and pathologic findings in anteroseptal infarction

Cited by 160 publications

References 29 publications

Quantitation of Variations in Regional Left Ventricular Function in Isolated Disease of the Left Anterior Descending Coronary Artery

Quantitation of Variations in Regional Left Ventricular Function in Isolated Disease of the Left Anterior Descending Coronary Artery

Can We Differentiate by the Admission Electrocardiogram between Anterior Wall Acute Myocardial Infarction due to a Left Anterior Descending Artery Occlusion Proximal to the Origin of the First Septal Branch and a Postseptal Occlusion?

Accuracy of various techniques in the measurement of the duration of the q wave: A possible source of error in diagnosing myocardial infarction by electrocardiography

Contact Info

Product

Resources

About