I Diretriz Brasileira de Miocardites e Pericardites

Montera, Marcelo Westerlund; Mesquita, Evandro Tinoco; Colafranceschi, Alexandre Siciliano; Oliveira, Amarino Carvalho de; Rabischoffsky, Arnaldo; Ianni, Bárbara Maria; Rochitte, Carlos Eduardo; Mady, Charles; Mesquita, Cláudio Tinoco; Azevedo, Clerio F.; Bocchi, Edimar Alcides; Saad, Eduardo; Marcondes‐Braga, Fabiana G.; Fernandes, Fábio; Ramires, Felix José Alvarez; Bacal, Fernando; Feitosa, Gílson Soares; Figueira, Hélio Roque; Neto, João David de Souza; Moura, Lídia Ana Zytynski; Campos, Luiz Antonio de Almeida; Bittencourt, Marcelo Imbroinise; Barbosa, Márcia de Melo; Moreira, Maria da Consolação Vieira; Higuchi, Maria de Lourdes; Schwartzmann, Pedro Vellosa; Rocha, Ricardo Mourilhe; Pereira, Sabrina Bernárdez; Mangini, Sandrigo; Martins, Sílvia Marinho; Bordignon, Solange; Salles, Vitor Agueda

doi:10.5935/abc.2013s004

Cited by 56 publications

(76 citation statements)

References 234 publications

(286 reference statements)

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“…In the ICAP trial, 11 the treatment strategy with NSAIDs was not specified in advance but patients received treatment with NSAIDs for 7 to 10 days, followed by weaning. 1,2,6,18 Most patients have a good initial response to NSAID drugs associated with colchicine, and become asymptomatic within a few days. Patients with no or with only limited effusion, and adherent to NSAID-colchicine treatment can be treated as outpatients.…”

Section: 14mentioning

confidence: 99%

“…Weaning should be done every 5-10 mg, every 2 weeks, for patients with daily use of 25 to 50 mg of prednisone; for those using a dose of 15 to 25 mg of prednisone per day, weaning should be done every 2.5 mg, every 2 to 4 weeks; and for patients taking daily doses lower than 15 mg of prednisone, it is recommended weaning from 1-2.5 mg, every 2 to 6 weeks. 1,6,10 recurrent pericarditis…”

Section: 14mentioning

confidence: 99%

“…5 In Brazil, there are no official epidemiological data on pericardial impairment. 6 Approximately one third of cases of idiopathic pericarditis are associated with myocarditis, which is manifested by elevation of myocardial injury biomarkers, such as troponin I. 7 Left ventricular dysfunction and heart failure are rare, and clinical presentation with arrhythmias is uncommon in patients with myocarditis.…”

Section: Clinical Definitionmentioning

confidence: 99%

“…• Computed tomography and cardiac magnetic resonance (class IIa recommendation, level of evidence B): 6 these have good sensitivity for detection of pericardial effusion, assessing the thickness of the pericardium and myocardial impairment. The most sensitive method to detect acute pericarditis is delayed enhancement using MRI.…”

Section: Diagnosismentioning

confidence: 99%

“…A low-volume but quickly accumulated effusion can cause cardiac tamponade without extending the silhouette, which reinforces the importance of echocardiography in patients with acute pericarditis, even if the cardiac silhouette is normal to the X-ray. 1 • Echocardiogram (class I recommendation, level of evidence B): 6 this is important to detect the presence of pericardial effusion, signs of tamponade (Figures 2 and 3) or altered segmental contractility. It is indicated in all cases involving diagnostic uncertainty or signs of hemodynamic impairment.…”

Section: Diagnosismentioning

confidence: 99%

See 4 more Smart Citations

Acute pericarditis

Tonini

Melo²,

Fernandes³

2015

Rev. Assoc. Med. Bras.

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Conflict of interest: noneAcute pericarditis is a common disease caused by inflammation of the pericardium, usually benign and self-limited and can occur as an isolated or as a manifestation of a systemic disease entity. Represents 5% of all causes of chest pain in the emergency room. The main etiology are viral infections, although it can also be secondary to systemic diseases and infections. The main complication of acute pericarditis is pericardial effusion, triggering a cardiac tamponade. The first line of treatment is the use of anti-inflammatory and or acetylsalicylic acid. Most patients have a good initial response to an NSAID associated to colchicine and became asymptomatic within a few days. This review article seeks to contemplate the main clinical findings and armed investigation to optimize the diagnosis of this important disease, as well as addressing their therapeutic management.Keywords: acute pericarditis, pericardial effusion, chest pain, colchicine. clinical definitionThe pericardium is a fibroelastic sac composed of two layers (visceral and parietal), separated by a virtual space called the pericardial cavity, which is filled with 15 to 50 mL of plasma ultrafiltrate in healthy subjects. Acute pericarditis is a common disease caused by inflammation of the pericardium, usually benign and selflimited, and may occur as an isolated entity or as a manifestation of a systemic disease.The real incidence and prevalence of pericarditis is difficult to quantify. Necropsy studies suggest a prevalence of approximately 1%. It affects mainly young males (aged between twenty and fifty years) without previous pathologies and represents 5% of all causes of chest pain in the emergency room. In developed countries, about 80-90% of cases are idiopathic, assuming that its main etiology is viral infection, although it can also be secondary to systemic diseases and infections.

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