Identification of risk groups for oropharyngeal dysphagia in hospitalized patients in a university hospital

Bassi, Daiane; Furkim, Ana Maria; Silva, Cristiane Alves; Coelho, Mara Sérgia Pacheco Honório; Rolim, Maria Rita Pimenta; Alencar, Maria Luiza Aires de; Machado, Marcos José

doi:10.1590/s2317-17822014000100004

Cited by 35 publications

(49 citation statements)

References 40 publications

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“…In this study, /m/, /t/, and /g/ were used as the speech stimuli in different frequencies and it was found that the impact of these speech stimuli did not differ significantly in generating the P1 latency. A few studies showed that different stimulus types, such as pure tone, click, a speech stimulus used in generating P1 did not change the P1 latency [18,[22][23][24][25]. In this study, the P1 latency values were combined so that it was possible to analyse more data at the same time.…”

Section: Discussionmentioning

confidence: 98%

Changes in P1 latencies of children with normal hearing and those with cochlear implants

Eskicioğlu¹,

Kırkım²,

Gürkan³

et al. 2020

Turk J Med Sci

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The aim of this study was to determine the age-related latency interval of P1 latencies of children with normal hearing, and to evaluate the P1 latency changes after surgery in children who underwent cochlear implantation. Materials and methods: We evaluated 60 children with normal hearing and 16 children with cochlear implants aged 0-6 years using cortical auditory evoked potentials. P1 latencies were measured only once in the children with normal hearing, and on the postoperative first day, and the first, third, and sixth postoperative months in the children with cochlear implants. Results: There was a statistically significant decrease in the P1 latencies as the age increased in children with normal hearing (P < 0.001). It was determined that when the external partof the cochlear implant was applied, the P1 latencies of children with cochlear implants were significantly longer than those of age-matched children with normal hearing (P < 0.001). This difference disappeared in 10 children with implants at the third and sixth months, but significant differences remained in 6 children. Conclusion: P1 latency could be used as an objective tool to evaluate the normal development of auditory pathways, and may be helpful in the effective programming of children undergoing cochlear implantation.

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Section: Discussionmentioning

confidence: 98%

Changes in P1 latencies of children with normal hearing and those with cochlear implants

Eskicioğlu¹,

Kırkım²,

Gürkan³

et al. 2020

Turk J Med Sci

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“…Studies have found that while the P1 component can be recorded in all children, the P2 response can only be reliably detected at $ 2 years old. Additionally, chronological age has been found to be the most influential variable in the devel-opment of auditory pathways, since the latency of CAEP components appear to decrease in direct proportion to the age of the subject 4,5,7,8,27 . As shown in ►Table 1, all 19 participants in the present study displayed the P1/N1 components, while 17 displayed the P2/N2 component at 2 years old.…”

Section: Discussionmentioning

confidence: 99%

“…3 In neonates and infants, the study of AEPs contributes to the assessment of the sensitivity, maturation, and neuroplasticity of auditory pathways, consisting an important source of information on auditory processing and the potential need of sound amplification, facilitating the implementation of early intervention programs, if necessary. [4][5][6][7][8] Cortical auditory evoked potentials (CAEPs) are longlatency responses also known as exogenous potentials, since they are determined by the physical properties of the stimulus rather than by the response of the subject. Cortical…”

Section: Introductionmentioning

confidence: 99%

“…17 There is no consensus in the literature regarding the age at which different components of LLAEPs first appear, or the age-based reference values for signal latency, 17 although studies on the topic have been conducted. 4,5,[7][8][9]11,12,14,[18][19][20][21][22][23][24][25][26][27][28][29][30][31] To date, no studies appear to have investigated CAEPs using verbal stimuli and the Smart EP platform (Intelligent Hearing Systems, Miami, FL, USA) in 2-year-old subjects. We hypothesized that P2 and N2 waves would be present by the age of 2 years old, since the level of myelination of the auditory cortex at this age would allow for the emergence of these components of the CAEP.…”

Section: Introductionmentioning

confidence: 99%

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Cortical Auditory Evoked Potentials in 2-Year-Old Subjects

Rechia

D'Agostini

Sousa

et al. 2019

Int Arch Otorhinolaryngol

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Introduction Cortical auditory evoked potentials (CAEPs) can be used to evaluate both peripheral and cortical components of auditory function, and contribute to the assessment of functional sensitivity and auditory thresholds, especially in neonates and infants. Auditory evoked potentials reflect auditory maturity and precede the acquisition of more complex auditory and cognitive skills, and are therefore crucial for speech and language development. Objective The aim of the present study was to determine the presence, latency and amplitude of CAEP components in response to verbal stimuli in children aged 2 years old. Methods The sample consisted of 19 subjects, 10 of whom were male while 9 were female. All of the participants were 24 months old at the time of assessment. Results A total of 17 of the participants displayed all components of the CAEP. Additionally, no significant differences were observed between genders or ears in the present sample. The presence of all components of the CAEP in subjects aged 2 years old confirms the existence of a critical period for the maturation of auditory pathways in the first 2 years of life. Conclusion In the present study, in addition to the P1/N1 components, it was possible to observe the presence of the CAEP P2/N2 components in individuals aged 24 months, confirming the existence of a critical period for the maturation of the auditory pathways in the first 2 years of life.

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“…Respiratory diseases, chronic obstructive pulmonary disease, and xerostomia, were indicated as risk factors for oropharyngeal dysphagia [3]. Another retrospective study reported a high incidence of risk for oropharyngeal dysphagia in hospitalized patients, and hospitalized patients with CHF.…”

Section: Introductionmentioning

confidence: 99%

Cognitive Dysfunction and Malnutrition Are Independent Predictor of Dysphagia in Patients with Acute Exacerbation of Congestive Heart Failure

et al. 2016

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Early detection and intervention for dysphagia is important in patients with congestive heart failure (CHF). However, previous studies have focused on how many patients with dysphagia develop CHF. Studies focusing on the comorbidity of dysphagia in patients with CHF are rare. Additionally, risk factors for dysphagia in patients with CHF are unclear. Thus, the aim of this study was to clarify risk factors for dysphagia in patients with acute exacerbation of CHF. A total of 105 patients, who were admitted with acute exacerbation of CHF, were enrolled. Clinical interviews, blood chemistry analysis, electrocardiography, echocardiography, Mini-Mental State Examination (MMSE), exercise tolerance tests, phonatory function tests, and evaluation of activities of daily living (ADL) and nutrition were conducted on admission. After attending physicians permitted the drinking of water, swallowing screening tests were performed. Patients were divided into a dysphagia group (DG) or a non-dysphagia group (non-DG) based on Functional Oral Intake Scale level. Among the 105 patients, 38 had dysphagia. A greater number of patients had history of aspiration pneumonia and dementia, and there was a higher age, N-terminal pro-B-type natriuretic peptide level in the DG compared with the non-DG. MMSE scores, exercise tolerance, phonatory function, status of ADL, nutrition, albumin, and transthyretin were lower in the DG compared with the non-DG. In multivariate analysis, after adjusting for age and sex, MMSE, BI score, and transthyretin was independently associated with dysphagia. Comorbidity of dysphagia was 36.1% in patients with acute exacerbation of CHF, and cognitive dysfunction and malnutrition may be an independent predictor of dysphagia.

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Identification of risk groups for oropharyngeal dysphagia in hospitalized patients in a university hospital

Cited by 35 publications

References 40 publications

Changes in P1 latencies of children with normal hearing and those with cochlear implants

Changes in P1 latencies of children with normal hearing and those with cochlear implants

Cortical Auditory Evoked Potentials in 2-Year-Old Subjects

Cognitive Dysfunction and Malnutrition Are Independent Predictor of Dysphagia in Patients with Acute Exacerbation of Congestive Heart Failure

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