IgA-dominant infection-related glomerulonephritis with NAPlr-positive tubulointerstitial nephritis

Okunaga, Issei; Makino, Shin-ichi; Honda, Daiske; Tatsumoto, Narihito; Aizawa, Masashi; Oda, Takashi; Asanuma, Katsuhiko

doi:10.1007/s13730-023-00782-x

Cited by 2 publications

(1 citation statement)

References 25 publications

Supporting

Mentioning

Contrasting

Order By: Relevance

“…[ 46 , 47 ]. Moreover, most of these pathogenic strains (especially E. coli , P. aeruginosa , and S. aureus ) enter the bloodstream and spread to various organs including kidneys causing infections (through invasion and/or toxin elaboration) like urinary tract infections (UTIs), catheter-associated urinary tract infections (CAUTIs), cystitis, pyelonephritis and emphysematous pyelonephritis, glomerulonephritis, tubulointerstitial nephritis, kidney/renal abscesses, or sepsis in critical cases [ 48 , 49 , 50 , 51 , 52 , 53 , 54 ].…”

Section: Gut Dysbiosis Uremic Toxicity and Key Kidney-associated Dise...mentioning

confidence: 99%

Biotics (Pre-, Pro-, Post-) and Uremic Toxicity: Implications, Mechanisms, and Possible Therapies

Mitrea,

Medeleanu,

Pop

et al. 2023

Toxins

View full text Add to dashboard Cite

In recent years, more scientific data have pointed out the close connection between intestinal microbial community, nutritional habits, lifestyle, and the appearance of various affections located at certain anatomical systems. Gut dysbiosis enhances the formation and accumulation of specific metabolites with toxic potential that induce the appearance of kidney-associated illnesses. Intestinal microbes are involved in the degradation of food, drugs, or other ingested products that lead to the formation of various metabolites that end up in renal tissue. Over the last few years, the possibilities of modulating the gut microbiota for the biosynthesis of targeted compounds with bioactive properties for reducing the risk of chronic illness development were investigated. In this regard, the present narrative review provides an overview of the scientific literature across the last decade considering the relationship between bioactive compounds, pre-, pro-, and post-biotics, uremic toxicity, and kidney-associated affections, and the possibility of alleviating the accumulation and the negative effects of uremic toxins into the renal system.

show abstract

Section: Gut Dysbiosis Uremic Toxicity and Key Kidney-associated Dise...mentioning

confidence: 99%

Biotics (Pre-, Pro-, Post-) and Uremic Toxicity: Implications, Mechanisms, and Possible Therapies

Mitrea,

Medeleanu,

Pop

et al. 2023

Toxins

View full text Add to dashboard Cite

show abstract

Comments and suggestions to the article, titled “IgA‑dominant infection‑related glomerulonephritis with NAPlr‑positive tubulointerstitial nephritis”

Taniguchi

2023

CEN Case Rep

View full text Add to dashboard Cite

IgA-dominant infection-related glomerulonephritis with NAPlr-positive tubulointerstitial nephritis

Cited by 2 publications

References 25 publications

Biotics (Pre-, Pro-, Post-) and Uremic Toxicity: Implications, Mechanisms, and Possible Therapies

Biotics (Pre-, Pro-, Post-) and Uremic Toxicity: Implications, Mechanisms, and Possible Therapies

Comments and suggestions to the article, titled “IgA‑dominant infection‑related glomerulonephritis with NAPlr‑positive tubulointerstitial nephritis”

Contact Info

Product

Resources

About