“…Intracranial circulation in the involved internal carotid artery territory is supplied by the contralateral carotid artery and from the vertebrobasilar circulation through the anterior and posterior communicating arteries, respectively 2 . Complete absence of bony carotid canal helps to differentiate agenesis from aplasia or hypoplasia 3 . Although most AICA patients remain asymptomatic, they may present with headache, cerebral infarcts, transient ischemic attacks, epilepsy, intracranial aneurysm, and amaurosis fugax 1,4 .…”