Imitators of epileptic seizures

Panayiotopoulos, C. P.

doi:10.1007/978-1-4471-4023-8

2012

DOI: 10.1007/978-1-4471-4023-8

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Imitators of epileptic seizures

C. P. Panayiotopoulos¹

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2014

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Cited by 2 publications

References 165 publications

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Diagnostic Role of ECG Recording Simultaneously With EEG Testing

Kendirli

Aparcı

Kendirli³

et al. 2014

Clin EEG Neurosci

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Arrhythmia is not uncommon in the etiology of syncope which mimics epilepsy. Data about the epilepsy induced vagal tonus abnormalities have being increasingly reported. So we aimed to evaluate what a neurologist may gain by a simultaneous electrocardiogram (ECG) and electroencephalogram (EEG) recording in the patients who underwent EEG testing due to prediagnosis of epilepsy. We retrospectively evaluated and detected ECG abnormalities in 68 (18%) of 376 patients who underwent EEG testing. A minimum of 20 of minutes artifact-free recording were required for each patient. Standard 1-channel ECG was simultaneously recorded in conjunction with the EEG. In all, 28% of females and 14% of males had ECG abnormalities. Females (mean age 49 years, range 18-88 years) were older compared with the male group (mean age 28 years, range 16-83 years). Atrial fibrillation was more frequent in female group whereas bradycardia and respiratory sinus arrhythmia was higher in male group. One case had been detected a critical asystole indicating sick sinus syndrome in the female group and treated with a pacemaker implantation in the following period. Simultaneous ECG recording in conjunction with EEG testing is a clinical prerequisite to detect and to clarify the coexisting ECG and EEG abnormalities and their clinical relevance. Potentially rare lethal causes of syncope that mimic seizure or those that could cause resistance to antiepileptic therapy could effectively be distinguished by detecting ECG abnormalities coinciding with the signs and abnormalities during EEG recording.

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Diagnostic Role of ECG Recording Simultaneously With EEG Testing

Kendirli

Aparcı

Kendirli³

et al. 2014

Clin EEG Neurosci

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Novel GLRB Gene Mutation in a Saudi Baby with Hyperekplexia

Rizk¹

2014

INDJ

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Aim:We aim to describe a case of hyperekplexia in a Saudi neonate due to Novel mutation in GLRB. Case Presentation: One month old Saudi neonate with hypertonicity, repetitive episodes of jitteriness and exaggerated startle reflex. Discussion: Hyperekplexia (OMIM:149400, 138492 & 604159) is considered a rare, autosomal dominant neurological disorder that presents early in life with hypertonicity, exaggerated startle response and life threatening neonatal apnea. It has been caused by mutation in the alpha-1subunit (GLRA1) on chromosome 5q32, Beta subunit (GLRB) gene on chromosome 4q31 of the inhibitory glycine receptor and GLYT2 gene (SLC6A5) on chromosome 11p15 which encodes a presynaptic glycine transporter. Conclusion: Raising awareness of the presence of this treatable disease may prevent unnecessary exposure to anti-epileptic medications, prevent life threatening apneas and improve long term outcome.

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Imitators of epileptic seizures

Cited by 2 publications

References 165 publications

Diagnostic Role of ECG Recording Simultaneously With EEG Testing

Diagnostic Role of ECG Recording Simultaneously With EEG Testing

Novel GLRB Gene Mutation in a Saudi Baby with Hyperekplexia

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