“…2 Takeshita et al 24 suggested that coronary collaterals develop in response to intermittent myocardial ischemia and that these collaterals are preserved even if they are closed at rest, in order to offer immediately function on acute coronary artery occlusion, after recruitment. Indeed, Herlitz et al 25 showed that patients with chronic angina pectoris (AP) before an acute MI had smaller infarcts compared with patients with AP of short duration before an acute MI.…”
Section: Determinants Of Coronary Collateral Circulation Myocardial Imentioning
“…2 Takeshita et al 24 suggested that coronary collaterals develop in response to intermittent myocardial ischemia and that these collaterals are preserved even if they are closed at rest, in order to offer immediately function on acute coronary artery occlusion, after recruitment. Indeed, Herlitz et al 25 showed that patients with chronic angina pectoris (AP) before an acute MI had smaller infarcts compared with patients with AP of short duration before an acute MI.…”
Section: Determinants Of Coronary Collateral Circulation Myocardial Imentioning
“…during angiography). They can, however, can be rendered visible if contrast is injected into the contralateral artery during coronary spasm [ 388 ] or during temporary balloon occlusion of the recipient artery [ 389 ]. These collaterals have been termed "recruitable" [ 390 ].…”
Section: Visible and Recruitable Coronary Collateralsmentioning
“…ST-segment elevation was caused by a transient occlusion of the major coronary artery, whereas ST-segment depression was caused by incomplete occlusion of coronary branches and invariably associated with the extensive coronary artery disease and rich collateral networks. Coronary collaterals develop with or without coronary artery disease (Yasue et al 1981;Takeshita et al 1982) and can modify the extent and severity of myocardial ischemia.…”
Section: Coronary Artery Spasm and Coronary Ischemic Syndromesmentioning
confidence: 99%
“…However, coronary spasm is the cause of postinfarction angina in a subset of patients with cyclic ST elevation. These patients may have no critical coronary stenosis on angiography Koiwaya et al 1982) and be effectively treated with calcium channel blockers (CCBs). The prognosis is generally favorable with CCBs in this population Koiwaya et al 1982).…”
Section: Coronary Artery Spasm and Coronary Ischemic Syndromesmentioning
Vasospastic angina (VSA) is one of the important functional cardiac disorders characterized by transient myocardial ischemia due to epicardial coronary artery spasm. In this chapter, I review the clinical features of VSA including pathophysiology, diagnosis, treatments, and prognosis. Furthermore, I also refer to the evidence of microvascular angina.
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