Impact of mobile intensive care units on STEMI delays and outcomes–Is it simply a matter of time?

Coiro, Stefano; Cavallini, Claudio

doi:10.1016/j.ejim.2020.01.022

Cited by 3 publications

(2 citation statements)

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“…Greater incidence of procedural cardiac complications, including MI, elevated creatine kinase, hypotension and cardiac arrest in CA-AKI patients may be the reason [ 32 ]. In addition, the total ischemic time didn’t show statistics difference between the two hydration groups and between the CA-AKI group and no CA-AKI group, which had been proved to be associated with mortality in patients with STEMI undergoing pPCI [ 11 ]. To sum up, although there was no statistical difference, the modified hydration relatively reduced the incidence of CA-AKI and would be a benefit to the prognosis in STEMI patients undergoing pPCI.…”

Section: Discussionmentioning

confidence: 99%

“…Patients’ demographic information, medical and medication history, and laboratory measurements were collected and confirmed through electronic medical records. The total ischemic time (the time from symptom initiation to reperfusion) was also collected [ 11 ]. The left ventricular ejection fraction (LVEF) was determined using 2-dimensional echocardiography during the index hospitalization.…”

Section: Methodsmentioning

confidence: 99%

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Role of modified hydration for preventing contrast-associated acute kidney injury in patients with ST-segment elevation myocardial infarction after primary percutaneous coronary intervention

Liu

Zhou

et al. 2022

Intern Emerg Med

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To assess the efficacy of modified hydration on contrast-associated acute kidney injury (CA-AKI) in ST-segment elevation myocardial infarction (STEMI) after primary percutaneous coronary intervention (pPCI). A total of 438 patients were randomly assigned to 2 groups. The traditional hydration group (group I) was given at a rate of 1 ml/kg/h for 24 h, and the modified hydration group (group II) was given at a rate of 3 ml/kg/h in the first 4 h, and then reduced to 1 ml/kg/h for 12 h. 0.3 mg/kg of furosemide was given 1-h after hydration. The primary endpoint was the incidence of CA-AKI, and the secondary endpoint was the incidence of major adverse cardiovascular events (MACEs) during a median of 22.4 months (IQR 9.6, 32.6 months) follow-up. The incidence of CA-AKI was 8.7%. Among these, Group I was 9.1% and group II was 8.2%, respectively. There was no significant difference in CA-AKI and creatinine levels between the two hydration groups. Multivariable logistics regression analysis revealed that creatinine, white blood cells, and N-terminal pro-B-type natriuretic peptide were associated with CA-AKI. Moreover, CA-AKI was an independent predictor for all-cause death and cardiac death during the follow-up period. The modified hydration may reduce the incidence of CA-AKI, although this difference was not statistically significant. The relationship between CA-AKI and mortality strengthened as creatinine times above baseline increased. Mitigating the occurrence of CA-AKI may reduce all-cause death and cardiac death.

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Section: Discussionmentioning

confidence: 99%

Section: Methodsmentioning

confidence: 99%