“…Structural and functional changes related to advanced HF, such as progressive myocardial fibrosis, adrenergic hyperactivity, mechanical and electrical remodelling, and metabolic dysregulation, all contribute to the genesis and maintenance of VT, which may occur in up to 30 % of these patients. 5 Moreover, indicators of advanced HF, such as very low ejection fraction and advanced New York Heart Association (NYHA) functional class, have been associated with an increased rate of periprocedural complications, VT recurrence and mortality in patients undergoing CA of VT. [6][7][8][9] Ventricular arrhythmias (VAs) may worsen HF status, increasing mortality and hospitalisations. 5,7 In this setting, pre-procedural risk stratification to identify high-risk patients can allow for preprocedural planning and optimisation of overall clinical status before the procedure, improving patient safety and post-procedural outcomes.…”