“…1 ± 3 The heightened sympathetic activity during an episode of autonomic hyperre¯exia accounts for several of the clinical features commonly observed including sudomotor and pilomotor phenomenon, 1,4 ± 6 vasomotor sequelae, 1 ± 4,7 and alterations in cardiac inotropic and chronotropic activity. 1,2,4,6,7 Cardiac abnormalities described in association with episodes of autonomic hyperre¯exia include bradycardia 1,4,6,7 and tachycardia, 2,6,7 premature atrial and ventricular contractions, 1,8 atrial ®brillation 9,10 and conduction block. 8 Despite the occurrence of these arrhythmias associated with autonomic hyperre¯exia, they are seldom recognized as a cause of cardiac arrest in individuals with traumatic spinal cord injury.…”