Increased P wave dispersion after the radiofrequency catheter ablation in overt pre-excitation patients: the role of atrial vulnerability

Risk factors responsible for atrial fibrillation development between symptomatic patients with concealed or manifest atrioventricular accessory pathways

“…Therefore, P wave duration should be measured after the ablation of APs. Previous study reported that Pmax and Pd became significantly longer in patients with AF history even when left atrial diameter and left ventricular ejection fraction were similar between AF and the control groups [23]. It suggested that Pmax and Pd might be more sensitive than left atrial diameter in reflecting atrial remodeling during AF genesis.…”

“…Although with some inconsistencies, the majority of previous studies observed that AAP ERP appeared shorter in AF group than in the control group for patients with manifest APs [8], [23], [24], [25]. Soylu et al [23] reported that, both AAP ERP and RAP ERP were shorter in AF group.…”

“…Soylu et al [23] reported that, both AAP ERP and RAP ERP were shorter in AF group. Fujimura et al [24] reported that AAP ERP was shorter in AF group while RAP ERP remained similar between AF group and the control.…”

Association of P wave duration and dispersion with the risk for atrial fibrillation: Practical considerations in the setting of coronary artery disease

“…An increase in PWD indicates heterogeneous intraatrial and interatrial conduction and discontinuous anisotropic propagation of sinus impulses, providing a substrate that favors reentry mechanisms [3]. Prolonged Pmax and increased PWD have been suggested to represent independent predictors for atrial fibrillation (AF) in patients with secundum atrial septal defect [4], with hypertrophic cardiomyopathy [5], with hyperthyroidism [6], with rheumatic mitral stenosis [7], with isolated coronary artery ectasia [8], with obstructive sleep apnea [9]; and in those after cardioversion [10] or after radiofrequency catheter ablation for overt pre-excitation [11]. The AF, whether paroxysmal or chronic, is the most common sustained arrhythmia encountered in clinical practice that produces substantial excess cardiovascular morbidity and mortality.…”

Atrial fibrillation with Wolff-Parkinson-White syndrome

“…13 Several mechanisms responsible for the development of AF in the WPW syndrome were investigated in detail, namely, the spontaneous degeneration of AVRT into AF, the electrical properties of the AP, the effects of AP on atrial architecture, and the intrinsic atrial muscle vulnerability. [14][15][16][17][18][19][20][21][22] Considering all the mechanisms proposed to explain the development of atrial fibrillation in the WPW syndrome, this review analizes the available data on this singular and interesting topic since AF has a reserved prognostic significance in patients with the WPW syndrome, and has an unusually high incidence in the absence of any clinical evidence of organic heart disease.…”