A 56-year-old man free from structural heart disease underwent radiofrequency (RF) catheter ablation of drugrefractory paroxysmal atrial flutter (AFL). A decapolar catheter was advanced with its proximal 9 and 10 poles placed in the proximal coronary sinus (CS), and a Halo deflectable duodecapolar catheter was positioned parallel to the tricuspid annulus (TA), across the inferior vena cava (IVC)-TA isthmus, with its tip at the CS ostium. During electrophysiologFigure 1. A: Surface electrocardiographic recording of the initiation of typical atrial flutter-like rhythm following atrial fibrillation induced after isthmus ablation. Typical flutter waves appeared following the 5th atrial fibrillatory cycle. B: Intracardiac electrograms during the AFL-like tachycardia. A regular atrial rhythm with a cycle length of 232 ms was recorded in a CCW activation sequence around the TA. Note the presence of apparent conduction delay between the atrial electrograms recorded at the adjacent Halo 7-8 and Halo 5-6 recording sites (bidirectional arrows), in the area of the ablated line shown in Figure 2A,B. Halo 21-22 to 1-2 = proximal to distal recording sites of the Halo catheter; HBE 3-4 and 1-2 = proximal and distal His-bundle electrograms; CS 8-9 to 1-2 = proximal and distal coronary sinus recordings.ical study, the clinical tachycardia developed spontaneously, following atrial fibrillation induced by bursts of rapid atrial pacing. On the 12-lead electrocardiogram recorded during the tachycardia the flutter wave was positive in leads II, III, aV F , V 5 , and V 6 , and negative in lead V 1 . No other atrial tachyarrhythmia was inducible by any standard method of induction. The atrial activation sequence along the TA during ongoing tachycardia, combined with entrainment pacing along the TA, confirmed the presence of typical clockwise (CW) AFL. Linear RF ablation blocked conduction across the IVC-TA isthmus, terminated the AFL and restored sinus rhythm. After confirmation of bidirectional block through the isthmus by differential pacing, a new tachycardia developed reproducibly following atrial fibrillation induced by bursts of rapid atrial pacing (Fig. 1A). On the 12-lead electrocardiogram recorded during the tachycardia, (a) the flutter wave was negative in leads II, III, aV F, V 5 , and V 6 , and positive in V 1 and V 2 (Fig. 1A), and (b) the atrial activation sequence along the TA during ongoing tachycardia was counterclockwise (CCW), similar to the recording during typical CCW