Posterior glenohumeral instability is an underappreciated cause of disability that may necessitate surgical intervention to allow functional glenoid restoration. However, posterior glenoid bone abnormalities, when sufficiently severe, may contribute to persistent instability despite a well-performed capsulolabral repair. Recognition and understanding of these lesions is critical to both surgical decision making and execution of the surgical plan. Numerous procedures have been described to address posterior instability including recent developments in arthroscopic grafting techniques. The purpose of this article was to provide an evidence-based strategy for diagnosis and management of posterior shoulder instability and glenoid bone loss.T o keep the humeral head centered within the glenoid during shoulder motion, the glenohumeral joint relies on the congruence of the glenoidhumeral head relationship, glenoid version, and a balance of static (ie, capsule, labrum, and glenohumeral ligaments) and dynamic (ie, rotator cuff musculature) stabilizers. When this balance becomes disrupted and concavity compression is altered, glenohumeral instability may occur, typically in an anterior or posterior direction.The primary soft-tissue stabilizers preventing posterior instability are the thick posterior-inferior glenohumeral ligament (PIGHL) and the posterior labrum. The PIGHL tightens in shoulder flexion to block posterior subluxation, while the posterior labrum centralizes the humeral head within the glenoid cavity and increases its functional depth. 1 Similar to their analogs in anterior instability, the posterior soft-tissue stabilizers are typically disrupted or attenuated. However, osseous stabilizers are often underappreciated for their importance in preventing recurrence. 2 Posterior instability and posterior capsular laxity most often occur secondary to repetitive posterior subluxation in the position of instability (ie, shoulder flexed and internally rotated) with the PIGHL under tension (eg, bench press and football blocking). It may also occur from acute injury including trauma and posterior muscular overpull. 3 Although posterior instability occurs with reported prevalence of 1.1 per 100,000 person-years, the true rate is likely higher. 4 In military populations, posterior instability is more commonly