Indocyanine Green Disappearance Enhanced by Prostaglandin E1in Patients with Hepatic Resection

Tsukada, Kazuhiro; Takeo, Satoshi; Aono, Takashi; Ishizuka, Dai; Fujita, Nobuhiro; Hatakeyama, Katsuyoshi

doi:10.1006/jsre.1996.0373

Cited by 9 publications

(3 citation statements)

References 4 publications

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“…In principle, the laser Doppler flow meter measures perfusion near the liver surface where arterial flow is dominant (25), so it is possible that the laser Doppler flow meter underestimated the liver blood flow after HAO. ICG clearance rates are influenced by the number of functioning liver cells and hepatic blood flow (26). Despite the marked differences in liver blood flow between the SMA group and other groups, there were no significant differences in ICG results.…”

Section: Survival Rate Of Animals and Histologic Examination Resultscontrasting

confidence: 64%

Efficacy of continuous infusion of prostaglandin E1 through the superior mesenteric artery against ischemic liver cell necrosis after hepatic artery occlusion

Kato

Sato²,

Kurokawa³

et al. 2003

Transplantation

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Section: Survival Rate Of Animals and Histologic Examination Resultscontrasting

confidence: 64%

Efficacy of continuous infusion of prostaglandin E1 through the superior mesenteric artery against ischemic liver cell necrosis after hepatic artery occlusion

Kato

Sato²,

Kurokawa³

et al. 2003

Transplantation

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“…ICG-PDR, however, is not exclusively a marker of cell function but even more blood flow, and short-term changes in ICG-PDR probably reflect changes in blood flow rather than hepatocellular function [34]. Several studies showed that ICG-PDR responds within 1-h intervals to changes in pharmacological treatment [34][35][36][37][38] and prone positioning [39].…”

Section: Indocyanine Green Clearancementioning

confidence: 99%

“…Several studies showed that ICG-PDR responds within 1-h intervals to changes in pharmacological treatment [34][35][36][37][38] and prone positioning [39].…”

Section: Indocyanine Green Clearancementioning

confidence: 99%

Assessing liver function

Sakka

2007

Current Opinion in Critical Care

220

167

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Induction of Heat Shock Response: Effect on the Rat Liver with Carbon Tetrachloride‐induced Fibrosis from Ischemia‐reperfusion Injury

et al. 1998

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The role of heat shock pretreatment in the induction of tolerance for ischemia-reperfusion injury was investigated in rat livers with fibrosis produced by carbon tetrachloride (CCI4) injected subcutaneously. The control group (group C, n = 56) received no pretreatment except anesthesia, and the heat shock group (group HS, n = 56) were exposed to heat shock (42 degrees C) for 15 minutes. After a 48-hour recovery all rats were subjected to 30 minutes of warm ischemia. Western blotting analysis was employed for heat shock protein (HSP) 72 detection. The adenine nucleotide levels in liver tissue and the liver enzyme levels in serum were measured before and after ischemic intervention (seven animals were used at each of six time point measurements in both groups). HSP72 was induced in group HS at greater intensity than in group C. The survival rate on postoperative day 7 in group C (3/14) was significantly poorer than that in group HS (14/14) (p < 0.01). The higher survival rate in group HS was accompanied by more rapid recovery of the adenosine triphosphate level and lower serum levels of liver enzymes after reperfusion (p < 0.01 vs. group C). Heat shock preconditioning induces HSP72 in the rat liver with fibrosis and provides significantly increased tolerance of warm-ischemia reperfusion injury.

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Indocyanine Green Disappearance Enhanced by Prostaglandin E1in Patients with Hepatic Resection

Cited by 9 publications

References 4 publications

Efficacy of continuous infusion of prostaglandin E1 through the superior mesenteric artery against ischemic liver cell necrosis after hepatic artery occlusion

Efficacy of continuous infusion of prostaglandin E1 through the superior mesenteric artery against ischemic liver cell necrosis after hepatic artery occlusion

Assessing liver function

Induction of Heat Shock Response: Effect on the Rat Liver with Carbon Tetrachloride‐induced Fibrosis from Ischemia‐reperfusion Injury

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