“…The principal reasons for induced intracranial hypertension are high flow rinsing (used to improve visibility during bleeding or to maintain access in collapsing ventricles ) and obstruction of the outflow channel by tissue debris , blood clots or kinking of outflow tubes. These increases in ICP must be detected as soon as possible to prevent severe complications such as cardiovascular instability (Fabregas et al, 2002, Handler et al, 1994 , herniation syndromes, retinal bleeding (Boogaarts et al, 2008 ;Hoving et al, 2009) and excessive fluid resorption (Kalmar et al, 2009). Aside from these unambiguous complications animal research showed that awakening without apparent neurological deficit does not preclude histological damage (Kalmar et al 2009).…”