Intérêt du scanner avec injection dans le syndrome de Fitz-Hugh-Curtis

Moigne, F. Le; Lamboley, J.-L.; Vitry, T.; Salamand, P; Milou, F.; Farthouat, P.

doi:10.1016/j.gcb.2009.09.002

Cited by 5 publications

(3 citation statements)

References 6 publications

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“…In 1930 and 1934, Curtis 1 ) and Fitz-Hugh 2 ) successively reported FHCS, and determined that N. gonorrhoeae is the major causative pathogen. In 1978, Muller-Schoop et al 8 ) reported that C. trachomatis was the new causative pathogen, while recent studies have demonstrated that C. trachomatis is the much more common pathogen implicated in FHCS 9 , 10 , 11 ) . You et al 12 ) and Woo et al 13 ) reported that C. trachomatis pathogens accounted for 89% and 86% of FHCS cases, respectively.…”

Section: Discussionmentioning

confidence: 99%

“…Acute fibrinous inflammation of the liver capsule surface was seen in the acute phase of FHCS, and MSCT examinations often show no obvious abnormalities. The FHCS pathological changes of the chronic phase were liver capsule fibrosis, hyaline degeneration, localized or widespread liver capsule thickening, and band adhesions between the liver surface and the abdominal wall 10 , 16 , 21 , 22 ) . MSCT scanning can display homogeneous or inhomogeneous thickening of the liver capsule with or without hepatic subcapsular effusion or pleural effusion, but it cannot display the disease extent or whether local fiber adhesions exist and to what degree.…”

Section: Discussionmentioning

confidence: 99%

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Fitz-Hugh-Curtis syndrome: clinical diagnostic value of dynamic enhanced MSCT

et al. 2015

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[Purpose] This study aimed to investigate the clinical diagnostic value of dynamic enhanced multislice computed tomography (MSCT) for Fitz-Hugh-Curtis syndrome (FHCS). [Subjects and Methods] This study retrospectively analyzed the clinical features and manifestations of scanning and dynamic enhanced MSCT in 19 patients with FHCS. [Results] MSCT scans showed different degrees of liver capsule thickness in the lesion area: seven cases of sub-capsular effusion and three cases with a small amount of pleural effusion; thickness of the liver capsular arterial phase showing significant enhancement in 17 cases, and slight enhancement in two; portal venous and delayed phase enhancement decreased with no clear boundary of the liver parenchyma; and adjacent hepatic parenchymal involvement in five cases, in which the arterial phase appeared to have patchy or triangular enhancement, and unclear portal vein and delayed phase imaging findings. MSCT revealed pelvic inflammatory disease in 14 cases, peritonitis in two, endometritis combined with bilateral ovarian abscesses in two, and a tube-ovarian abscess in one. [Conclusion] Dynamic enhanced MSCT can accurately display liver capsule lesions and possible pelvic inflammatory diseases related to FHCS, suggest the infection source, and have high application value for making early, accurate diagnoses and improved prognosis.

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Section: Discussionmentioning

confidence: 99%

Section: Discussionmentioning

confidence: 99%

Fitz-Hugh-Curtis syndrome: clinical diagnostic value of dynamic enhanced MSCT

et al. 2015

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“…In the literature, approximately 12-25% of pelvic inflammatory disease cases occur concomitantly with FHC. The frequently involved etiological agents are of bacterial origin, and N. gonorrhoeae and C. trachomatis are among the most common (2,6,7). These microorganisms can travel from the pelvis to infect the surface capsule of the liver or the spleen via the peritoneal fluid (4,8).…”

Section: Discussionmentioning

confidence: 99%