2008
DOI: 10.1007/s12262-008-0082-0
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Intestinal duplication

Abstract: Gastrointestinal duplications are rare but interesting clinical entities. They have a varied presentation, with most of them showing up in paediatric population. Clinical features may vary from asymptomatic abdominal masses to bowel obstruction or perforation. This review traces the embryological origin and describes the anatomical types of duplications. An outline of the principles of management is described.

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Cited by 14 publications
(27 citation statements)
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“…[ 2 ] Since, the first report by Calder in 1733 several case reports have been published. [ 3 ] However, there is a paucity of studies from the Indian subcontinent. Herein, we present our experience with 38 EDs treated between January 2003 and July 2014.…”
Section: Introductionmentioning
confidence: 99%
“…[ 2 ] Since, the first report by Calder in 1733 several case reports have been published. [ 3 ] However, there is a paucity of studies from the Indian subcontinent. Herein, we present our experience with 38 EDs treated between January 2003 and July 2014.…”
Section: Introductionmentioning
confidence: 99%
“…Gastrointestinal duplications are less common (1/4500 birth). Though many different terms were used before to determine this patologies like giant diverticula, inclusion cyst, enteric cyst, enterogenous cyst, reduplication, ileum duplex, unusual Meckel's diverticulum and accessory enteric formation, today we know all they are the same patology (7) . Usually they are tightly attached to the nearby digestive duct and located on the mesenteric border between the mesenteric leaves.…”
Section: Discussionmentioning
confidence: 99%
“… 2 These cysts can occur anywhere along the alimentary tract from the mouth to the anus, although the ileum is the most frequently involved region (35%) followed by the esophagus (19%), jejunum (10%), stomach (9%), and colon (7%). 15 CIDC is an extremely rare variant form of gastrointestinal duplication that has its own blood supply and does not communicate with the normal bowel segment.…”
Section: Discussionmentioning
confidence: 99%
“…Several theories were proposed to account for the development of enteric duplications, including recanalization after the solid epithelial stage of embryonic bowel development, persistent embryologic diverticula, and the intrauterine vascular accident theory. 15 , 16 , 17 Bremer's "aberrant vacuolization" theory suggests that epithelial proliferation occurs during embryonic intestine development and occludes the bowel lumen in the 6-week embryo ("solid stage"); thereafter, a vacuolization of the entire alimentary tract occurs, thus transforming the digestive system into a tube with a single lumen. Throughout the process of vacuole coalescence, an error might occur ("aberrant vacuolization"), resulting in the formation of 2 (or more) parallel channels that may or may not communicate with each other.…”
Section: Discussionmentioning
confidence: 99%