“…In this research, we found that while the pathology leading up to the procedure, the therapy, and the sedation used during the procedure were hypothesized to be in relation to the onset of PRES, in fact there does not seem to be a direct correlation with one pathology versus another. We have identified cases of postoperative PRES in patients treated for gastrointestinal pathology [27,30,47], gynecologic pathology [12], and even urologic pathology [31,48]. Several anesthesiologic reports [12,27,31,50] on PRES have argued that the major causative factor could be suboptimal pressure control in hypertensive patients [25,26].…”