Intraabdominal Pressure and Gastric Intramucosal pH: Is There an Association?

Sugrue, Michael; Jones, F.; Lee, Anna; Buist, Michael; Deane, S. A.; Bauman, Adrian; Hillman, Ken

doi:10.1007/s002689900149

Cited by 106 publications

(55 citation statements)

References 23 publications

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“…Raeburn and coworkers [2], for example, reported an incidence of 36% of ACS in trauma patients after damage control laparotomy. A considerable number of reports have demonstrated that IAH exerts deleterious effects on the cardiovascular system and on pulmonary, renal, and gastrointestinal function [3,4,5,7]. Almost 30% of trauma patients requiring damage control laparotomy who show ACS develop multiple organ failure.…”

Section: Introductionmentioning

confidence: 99%

Measurement of compartment pressure of the rectus sheath during intra-abdominal hypertension in rats

et al. 2006

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Section: Introductionmentioning

confidence: 99%

Measurement of compartment pressure of the rectus sheath during intra-abdominal hypertension in rats

et al. 2006

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“…When severe, tissue ischemia can result [32][33][34]. Clinically this can be monitored with gastric tonometry by recording the gastric mucosal pH, P CO 2 and GAP CO 2 :…”

Section: Gut Functionmentioning

confidence: 99%

Postinjury Primary Abdominal Compartment Syndrome

Balogh

Bendinelli

Pollitt

et al. 2008

Eur J Trauma Emerg Surg

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Postinjury abdominal compartment syndrome (ACS) has evolved during the 1980s together with the introduction of damage control surgery (DCS) principles. DCS made it possible to salvage severely injured trauma patients who previously would have exsanguinated due to uncontrollable coagulopathic bleeding. These patients had severe hemorrhagic shock; their abdomens were tightly packed and had ongoing massive resuscitation. ACS is a lethal complication of the damage control patients. For today the pathophysiological characteristics of ACS are described, the intra-abdominal pressure is measured on many intensive care units. Postinjury ACS (primary and secondary) is one of the better characterized etiological types of ACS: risk factors, diagnostic criteria, independent predictors and preventive strategies are all well documented. Since the mortality of full-blown postinjury ACS is still unacceptably high and does not seem to improve with earlier decompression, prevention is the recommended strategy to decrease the morbidity and mortality. Open abdomen is one of the important preventive strategies but it is not free from morbidity and mortality. With aggressive open abdomen management in postinjury ACS these complications can be minimized. More importantly, timely hemorrhage control and hemostatic resuscitation are the likely solutions for more efficient prevention of the postinjury ACS.

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“…In one study, 38% of patients admitted to an intensive care unit after major abdominal surgery had IAPs greater than 20 mm Hg. 19 The level of IAP at which the ACS occurs is variable. Clearly, variability in the physiologic response to a graded increase in pressure depends in part on the intravascular volume status of the patient as well as underlying pulmonary or renal dysfunction.…”

Section: Measurement Of Iapmentioning

confidence: 99%

Abdominal compartment syndrome

2007

Nursing

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© 1997 Canadian Medical Association (text and abstract/résumé)The abdominal compartment syndrome refers to the alterations in respiratory mechanics, hemodynamic parameters and renal function that occur as a result of a sustained increase in intra-abdominal pressure. The syndrome may follow a diverse series of insults, including laparotomy for severe abdominal trauma, ruptured abdominal aortic aneurysm and intra-abdominal infection. Diagnosis depends on recognizing the clinical picture in patients at risk, followed by an objective measurement of intra-abdominal pressure. Successful management may require abdominal decompression with temporary abdominal closure. Despite urgent decompression, the death rate is high because of the severity of the patients' underlying illness.Le syndrome du compartiment abdominal désigne les modifications de la mécanique respiratoire, des paramètres hémodynamiques et de la fonction rénale qu'entraîne une augmentation soutenue de la pression intra-abdominale. Le syndrome peut découler d'une série diverse d'atteintes, y compris une laparotomie à la suite d'un traumatisme abdominal grave, une rupture d'un anévrisme de l'aorte abdominale et une infection intra-abdominale. Le diagnostic dépend de l'identification du tableau clinique chez les patients à risque, suivie d'une mesure objective de la pression intra-abdominale. Un traitement réussi peut exiger une décompression abdominale avec fermeture temporaire de l'abdomen. Malgré les décompressions d'urgence, le taux de mortalité est élevé à cause de la gravité de la maladie sous-jacente des patients.

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Intraabdominal Pressure and Gastric Intramucosal pH: Is There an Association?

Abstract: This study evaluated the potential association between increased intraabdominal pressure (IAP) and abnormally low gastric intramucosal pH (pHi) (/= 20 mmHg and a pHi of Show more

Cited by 106 publications

References 23 publications

Measurement of compartment pressure of the rectus sheath during intra-abdominal hypertension in rats

Measurement of compartment pressure of the rectus sheath during intra-abdominal hypertension in rats

Postinjury Primary Abdominal Compartment Syndrome

Abdominal compartment syndrome

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