“…[10][11][12][13][14] Acquired webs occur secondary to endotracheal intubation, laryngeal trauma, laryngeal surgery near the anterior commissure, radiotherapy, and rarely, inflammatory processes, such as tuberculosis or diphtheria. 6,15,16 In the normal course of development, the laryngeal lumen is obliterated by the proliferation of the epithelial lamina in the sixth week of fetal life, which then recanalizes in the eighth to 10th week. Congenital laryngeal webs, subglottic stenosis, and laryngeal atresia result from various degrees of this recanalization failure 17 as a spectrum of congenital laryngeal anomalies.…”