Inventories for male and female sexual dysfunctions

Corona, Giovanni; Jannini, Emmanuele A.; Maggi, Mario

doi:10.1038/sj.ijir.3901410

Cited by 131 publications

(109 citation statements)

References 71 publications

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“…58 This clinical tool has the advantage of being standardized, easy to administer and score, and provides normal values in general and pathological populations. 59 Previous evidence linking FSD to obesity is very scanty. In 171 postmenopausal women, Kirchengast et al 56 reported that body weight and BMI were significantly related to the degree of reduced sexual interest.…”

Section: Obesity Metabolic Syndrome and Female Sexual Functionmentioning

confidence: 99%

Obesity and sexual dysfunction, male and female

et al. 2008

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Obesity has become a worldwide public health problem of epidemic proportions, as it may decrease life expectancy by 7 years at the age of 40 years: excess bodyweight is now the sixth most important risk factor contributing to the overall burden of disease worldwide. Overweight and obesity may increase the risk of erectile dysfunction (ED) by 30-90% as compared with normal weight subjects. On the other hand, subjects with ED tend to be heavier and with a greater waist than subjects without ED, and also are more likely to be hypertensive and hypercholesterolemic. The metabolic syndrome, characterized by a clustering of risk factors associated with insulin resistance and abdominal obesity, associates with ED. Moreover, women with the metabolic syndrome have an increased prevalence of sexual dysfunctions as compared with matched control women. Lifestyle changes aimed at reducing body weight and increasing physical activity induce amelioration of both erectile and endothelial functions in obese men. Moreover, preliminary evidence suggests that a Mediterranean-style diet might be effective in ameliorating sexual function in women with the metabolic syndrome. Lifestyle changes, mainly focussing on regular physical activity and a healthy diet, are effective and safe ways to reduce cardiovascular diseases and premature mortality in all population groups; they may also prevent and treat sexual dysfunctions in both sexes.

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Section: Obesity Metabolic Syndrome and Female Sexual Functionmentioning

confidence: 99%

Obesity and sexual dysfunction, male and female

et al. 2008

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“…9 This clinical tool has the advantage of being standardized, easy to administer and score and provides normal values in general and pathological populations. 10 To the best of our knowledge there are no reported studies assessing the relation between FSFI and body weight in women. Therefore, the aims of this study were: (a) to identify women with pathological values of FSFI across a wide range of body weight; (b) to compare them with control, age-matched women with normal values of sexual function and (c) to investigate the relationship between the amount and distribution of body fat and sexual function.…”

Section: Introductionmentioning

confidence: 99%

Association of body weight with sexual function in women

et al. 2007

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Sexual difficulties in women appear to be widespread in society; the relationship between female sexual function and obesity is unclear. This study aimed to investigate the relationship between body weight, the distribution of body fat and sexual function in women. Fifty-two, otherwise healthy women with abnormal values of female sexual function index (FSFI) score (p23) were compared with 66 control women (FSFI 423), matched for age and menopausal status. All women were free from diseases known to affect sexual function. FSFI strongly correlated with body mass index (BMI) (r ¼ -0.72, P ¼ 0.0001), but not with waist-to-hip ratio (r ¼ -0.09, P ¼ 0.48), in women with sexual dysfunction. Of the six sexual function parameters, desire and pain did not correlate with BMI, while arousal (r ¼ -0.75), lubrication (r ¼ -0.66), orgasm (r ¼ -0.56) and satisfaction (r ¼ -0.56, all Po0.001) did. FSFI score was significantly lower in overweight women as compared with normal weight women, while cholesterol and triglyceride levels were higher. On multivariate analysis, both age and BMI explained about 68% of FSFI variance, with a primacy of BMI over age (ratio 4:1). In conclusion, obesity affects several aspects of sexuality in otherwise healthy women with sexual dysfunction.

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“…A further advantage of the structured interview over the self-reported questionnaire is that a face-to-face interview facilitates a virtuous, intimate physician-patient relationship. 32 In fact, structured interviews have been found to have a greater diagnostic value than self-reported questionnaires in other contexts when involving possibly sensitive issues. 33 We have recently developed a brief (12 items) structured interview (ANDROTEST) specifically designed for the screening of hypogonadism (total T ,10.4 nmol l…”

Section: Structured Inventories For Detecting Lohmentioning

confidence: 99%

How to recognize late-onset hypogonadism in men with sexual dysfunction

Corona¹,

Rastrelli²,

Vignozzi³

et al. 2012

Asian J Androl

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Late-onset hypogonadism (LOH) has been considered the most common form of male hypogonadism with a prevalence of approximately 1 in 100 men. Diagnosis of LOH should be made in symptomatic men with unequivocally low serum testosterone (T) levels. However, its clinical presentation is often insidious and difficult to recognize because it is characterized by nonspecific symptoms that make differential diagnosis with physiological ageing problematic. Sexual dysfunction is the most important determinant for medical consultation and the most specific symptom associated with low T. We therefore analysed a consecutive series of 1734 subjects who attended our unit for sexual dysfunction to investigate the associations between low T (different thresholds), sexual parameters, medical history data (delayed puberty, pituitary disease or cryptorchidism) and their physical exam results. Metabolic parameters, in particular waist circumference, display the greatest accuracy in detecting low T. We found that only the association of several symptoms and signs could significantly raise the clinical suspicion of low T. Structured inventories, which cluster together symptoms and signs of hypogonadism, can help clinicians suspect androgen deficiency. In particular, structured interviews, such as ANDROTEST, have been demonstrated to have a greater accuracy when compared to self reported questionnaires in detecting low T levels. Keywords: late-onset hypogonadism (LOH); male hypogonadism; testosterone; sexual dysfunction INTRODUCTIONThe testis (the male gonad) continuously produces sex steroids (mostly testosterone (T)) and sperm soon after the onset of puberty. Testis activity is regulated by intratesticular factors (including T) and by extratesticular trophic factors released from the pituitary (i.e., gonadotropin luteinizing hormone (LH) and follicle-stimulating hormone (FSH)), which are strictly regulated by the hypothalamic peptide gonadotropin-releasing hormone (GnRH). Male hypogonadism is a failure in testis activity that is classically considered to be a partial or total communication breakdown among the hypothalamus, the pituitary and the testis itself. Current definitions of hypogonadism rely on T deficiency more than abnormal sperm production, but from a conceptual point of view, both factors contribute to testicular function and dysfunction.

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Inventories for male and female sexual dysfunctions

Cited by 131 publications

References 71 publications

Obesity and sexual dysfunction, male and female

Obesity and sexual dysfunction, male and female

Association of body weight with sexual function in women

How to recognize late-onset hypogonadism in men with sexual dysfunction

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