Ischaemic Stroke and Transient Ischaemic Attack

Ringleb, Peter A.; Bousser, Marie Germaine; Ford, Gary; Bath, Philip M.W.; Brainin, Michael; Caso, Valeria; Cervera, Álvaro; Chamorro, Ángel; Cordonnier, Charlotte; Csiba, László; Dávalos, Antoni; Diener, Hans‐Christoph; Ferro, José M.; Hacke, Werner; Hennerici, Michael G.; Kaste, Markku; Langhorne, Peter; Lees, Kennedy R.; Leys, Didier; Lodder, J.; Markus, Hugh S.; Mas, Jean Louis; Mattle, Heinrich; Muir, Keith W.; Norrving, Bo; Obach, Vı́ctor; Paolucci, Stefano; Ringelstein, E. Bernd; Schellinger, P. D.; Sivenius, Juhani; Skvortsova, Veronika; Sunnerhagen, Katharina Stibrant; Thomassen, Lars; Toni, Danilo; Kummer, Raquel; Wahlgren, Nils-Gunnar; Walker, Marion F; Wardlaw, Joanna M.

doi:10.1002/9781444328394.ch9

Cited by 32 publications

(34 citation statements)

References 541 publications

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“…Medical therapy with antiplatelet agents or anticoagulation versus surgical or percutaneous closure are the most widely discussed treatment options [17].…”

Section: Discussionmentioning

confidence: 99%

Patient with patent foramen ovale and thrombophilia, after ischemic stroke, acute coronary syndrome and pulmonary embolism (RCD code: IV-2B.O)

Komar

Stępniewski

Przewłocki

et al. 2015

Journal of Rare Cardiovascular Diseases

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Patent foramen ovale (PFO) is a residue of the foramen ovale, an integral part of the normal fetal circulation. It closes in most of newborns, however 25-30% of adults may have it patent. Conditions such as crytpogenic stroke, migraine and vascular headaches or decompression sickness are strongly related to the presence of PFO. Less frequently acute myocardial infarction, renal infarction or acute mesenteric ischemia may also occur. We present a case of a 67-year-old man after cerebral ischemic episode, acute myocardial infarction treated with primary coronary intervention and pulmonary embolism, who was eventually diagnosed with PFO and factor V Leiden mutation. Life-long anticoagulation was initiated in this patient as a standard-of-care. He additionally underwent successful percutaneous PFO closure. As a result no subsequent thromboembolic complication occurred in this patient at the follow-up period. JRCD 2015; 2 (2): 52-55

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“…Medical therapy with antiplatelet agents or anticoagulation versus surgical or percutaneous closure are the most widely discussed treatment options [17].…”

Section: Discussionmentioning

confidence: 99%

Patient with patent foramen ovale and thrombophilia, after ischemic stroke, acute coronary syndrome and pulmonary embolism (RCD code: IV-2B.O)

Komar

Stępniewski

Przewłocki

et al. 2015

Journal of Rare Cardiovascular Diseases

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“…Causes of late presentation of patients are inability to recognize of the symptoms of cerebrovascular event (SVE) by patients, ignoring symptoms, and status can't be determined due to unconsciousness induced SVE. 14 The causes of admission of our patient after about 10 h from the onset of his complaints were transportation difficulties and hope that his complaints will be resolved. As a result, stroke patients who present to the emergency department with a loss of vision and who have risk factors for thromboembolism should be evaluated more carefully and detailed neurological and visual examination should be performed.…”

mentioning

confidence: 83%

Acute bilateral vision loss in Emergency Department: A case report

Ecevit¹

2015

Tr J Emerg Med

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a b s t r a c tStroke occurs due to the interruption of blood flow to the brain and it is divided into ischemic and hemorrhagic. In the ischemic strokes, while the most commonly affected vessel is median cerebral artery (MCA), it is particularly affected bilateral posterior cerebral artery (PCA) is very rare condition. In this study, a case of sudden loss of vision and bilateral occipital infarct associated with bilateral vertebral system pathology and methylene tetrahydrofolate reductase (MTHFR) gene mutation were reported. A 62-year-old man was admitted with sudden loss of vision complaint starting 10 h before applying to emergency department. The patient was oriented and cooperative. On neurological examination, there was complete loss of vision in the right eye and only a response to light in the left eye. On the brain computerized tomography (CT), ischemic lesions were observed in the bilateral occipital areas and on magnetic resonance imaging (MRI), there were foci showing diffusion limitation in cortico-subcortical areas of bilateral parieto-occipital region. On the detailed examination at the clinic, MTHFR (a1298c) gene mutation was detected. Bilateral occipital infarction is rare and its diagnosis can be difficult because of its atypical symptoms. Therefore, occipital infarction should be suspected when the only sign is isolated vision loss in patients with risk factor for thromboembolism in their history and detailed visualneurological examination of these patients should be performed.

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“…International guidelines and approval criteria for use of intravenous thrombolysis exclude patients with stroke whose time of symptom onset is unknown [26]. The use of a time window for tPA eligibility arises from evidence gathered through thousands of patients showing the declining benefit of thrombolytic therapy with time, with the cutoff lying at around 4.5 h after stroke onset, beyond which time it appears that treatment no longer leads to an increased odds ratio for favorable outcome [27 • ].…”

Section: Fluid-attenuated Inversion Recovery (Flair) In Wake-up Strokementioning

confidence: 99%

MR Imaging for Acute Stroke

Fiebach

Galinović

2014

Curr Radiol Rep

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Cerebral imaging is essential to differentiate the pathologies behind acute stroke. Excluding hemorrhage with a native CT is a prerequisite before thrombolytic drugs can be administered. With stroke MRI, vessel occlusion, ischemia and infarction can be visualized. This detailed diagnostic information can facilitate the decision for or against thrombolysis. Analysis of FLAIR and diffusion-weighted images enables the identification of patients in an early time window after ictus and is currently used in randomized trials of wake-up stroke.

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Ischaemic Stroke and Transient Ischaemic Attack

Cited by 32 publications

References 541 publications

Patient with patent foramen ovale and thrombophilia, after ischemic stroke, acute coronary syndrome and pulmonary embolism (RCD code: IV-2B.O)

Patient with patent foramen ovale and thrombophilia, after ischemic stroke, acute coronary syndrome and pulmonary embolism (RCD code: IV-2B.O)

Acute bilateral vision loss in Emergency Department: A case report

MR Imaging for Acute Stroke

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