1994
DOI: 10.1016/s0021-5198(19)35748-8
|View full text |Cite
|
Sign up to set email alerts
|

K+-Linked Release of Oxidized Glutathione Induced by tert-Butyl Hydroperoxide in Perfused Rat Liver Is Independent of Lipid Peroxidation and Cell Death

Help me understand this report

Search citation statements

Order By: Relevance

Paper Sections

Select...
1

Citation Types

0
1
0

Year Published

2005
2005
2008
2008

Publication Types

Select...
2

Relationship

0
2

Authors

Journals

citations
Cited by 2 publications
(1 citation statement)
references
References 27 publications
0
1
0
Order By: Relevance
“…However, with a large influx of peroxides such as that occurring during the infusion of PN, reductase activity may be insufficient to maintain this ratio. In that case, GSSG is actively exported (43,44), resulting in hepatic glutathione being excreted in bile (44,45). Therefore, bile glutathione content may be an important marker in the induction of cholestasis, as it is a driving force in bile flow formation (34,35).…”
Section: Introductionmentioning
confidence: 99%
“…However, with a large influx of peroxides such as that occurring during the infusion of PN, reductase activity may be insufficient to maintain this ratio. In that case, GSSG is actively exported (43,44), resulting in hepatic glutathione being excreted in bile (44,45). Therefore, bile glutathione content may be an important marker in the induction of cholestasis, as it is a driving force in bile flow formation (34,35).…”
Section: Introductionmentioning
confidence: 99%