“…Despite the identification of the disease entity over 100 years ago, the etiopathogenesis of Kienböck disease is still uncertain. Even so, it has been associated with a variety of predisposing factors including single trauma, repetitive microtrauma, damage to the lunate blood supply, corticosteroid use, negative ulnar variance, decreased radial inclination, and lunate morphology [3][4][5]. Viegas et al classified the lunate morphology based on the absence (type I) or presence (type II) of hamate facet at least 2 mm.…”