Kinder-DIPS

Unnewehr, Suzan; Schneider, Silvia; Margraf, Jürgen

doi:10.1007/978-3-662-06607-2

Cited by 47 publications

(18 citation statements)

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“…All OCD probands fulfilled the diagnostic criteria for OCD according to DSM-IV [1]. Criteria for OCD were assessed by interviewing the children and parents with the respective versions of “Diagnostisches Interview bei psychischen Störungen im Kindes- und Jugendalter” (Kinder-DIPS) [21]. Severity and further characteristics of OCD were examined by interviewing the patients with the Children’s Yale-Brown Obsessive Compulsive Scale (CY-BOCS) (with a summary score above 16 points determined to be the cut-off for clinical impairment caused by OCD symptomatology) [16].…”

Section: Methodsmentioning

confidence: 99%

Trio study and meta-analysis support the association of genetic variation at the serotonin transporter with early-onset obsessive–compulsive disorder

Walitza

Marinova

Grünblatt

et al. 2014

Neuroscience Letters

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Despite compelling evidence for major genetic contributions to the etiology of obsessive-compulsive disorder (OCD), few genetic variants have been consistently associated with this debilitating illness. Molecular genetic studies in children and adolescents with OCD are of particular interest, since early onset of the disease has been observed to be associated with increased familiality. We replicate here for the first time in early-onset OCD patients, a previously reported association of OCD with the common gain-of-function LA allele at the serotonin transporter linked polymorphic region known as 5-HTTLPR in a collection of parent-offspring trios. The present meta-analysis of this recently refined serotonin transporter gene variant revealed further support for the LA allele conferring increased genetic susceptibility to OCD. We conclude that the 5-HTTLPR is currently the single best supported risk variant for OCD, in regards of early-onset OCD, albeit of modest effect size and the possibility that the conferred risk might not be specific to OCD.

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Section: Methodsmentioning

confidence: 99%

Trio study and meta-analysis support the association of genetic variation at the serotonin transporter with early-onset obsessive–compulsive disorder

Walitza

Marinova

Grünblatt

et al. 2014

Neuroscience Letters

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“…Internal consistency of the EDE-Q global score lies at α = .94 (N = 1354; Hilbert, de Zwaan, & Braehler, 2012) and at α = .86 (N = 352) for the global scale of the (Child) Eating Disorder Examination Questionnaire (Hilbert et al, 2013). Anxiety was measured with the 'State-Trait Anxiety Inventory' (Laux, Glanzmann, Schaffner, & Spielberger, 1981) or the equivalent children's version (Unnewehr, Joormann, Schneider, & Markgraf, 1992). Depression was examined by using the self-report questionnaire 'German depression inventory for children and adolescents' (Stiensmeier-Pelster, Schürmann, & Duda, 2000).…”

Section: Assessment Of General Psychopathologymentioning

confidence: 99%

Follow‐up Assessment of Cognitive Remediation Therapy in Adolescent Anorexia Nervosa: A Pilot Study

Herbrich

Noort

Pfeiffer

et al. 2017

Euro Eating Disorders Rev

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“…Additionally, obsessiveness was assessed with the corresponding subscale of the Derogatis Brief Symptom Inventory (BSI; Franke, 2000). Furthermore, the semistructured clinical interviews ('Kinder-DIPS'; Unnewehr, Schneider, & Margraf, 2008) that were performed showed that none of the AN patients fulfilled the diagnostic criteria for OCD or any other psychiatric disorder according to the DSM-IV or ICD-10, with the exception of a mild to moderate depressive episode. Additionally, perfectionism was evaluated by calculating the score of the corresponding subscale of the Eating Disorder Inventory (EDI-2; Thiel et al, 1997) self-report questionnaire.…”

Section: Participantsmentioning

confidence: 99%

Implicit sequence learning in juvenile anorexia nervosa: neural mechanisms and the impact of starvation

Firk

Mainz

Martin

et al. 2015

Child Psychology Psychiatry

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Our data suggest that the impaired implicit learning observed in adolescent AN patients before weight gain is a state-related dysfunction that normalises with weight gain. Thus, implicit learning deficits do not appear to represent a predisposition towards developing AN; rather, these deficits should be considered when planning psychotherapeutic interventions for acute AN. Reduced thalamic activation during the acute stage of AN may indicate a starvation-induced dysfunction of the neural circuitry that is involved in behavioural flexibility.

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Kinder-DIPS

Cited by 47 publications

References 0 publications

Trio study and meta-analysis support the association of genetic variation at the serotonin transporter with early-onset obsessive–compulsive disorder

Trio study and meta-analysis support the association of genetic variation at the serotonin transporter with early-onset obsessive–compulsive disorder

Follow‐up Assessment of Cognitive Remediation Therapy in Adolescent Anorexia Nervosa: A Pilot Study

Implicit sequence learning in juvenile anorexia nervosa: neural mechanisms and the impact of starvation

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