Korneale Dellen bei konjunktivaler Chemose im Anschluss an eine transkonjunktivale Orbitadekompression

Moesen, Ingemarie; Hafezi, Farhad; Paridaens, Dion

doi:10.1055/s-2007-963618

Cited by 6 publications

(6 citation statements)

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“…Most common ophthalmic causes of conjunctival chemosis include orbital trauma [2], orbital pseudotumor [3], scleritis [4], and transconjunctival orbital decompression [5]. To the best of our knowledge, our report is the first to describe prominent conjunctival chemosis as a presenting clinical feature of SO.…”

Section: Discussionmentioning

confidence: 98%

Sympathetic ophthalmia manifesting with conjunctival chemosis

et al. 2009

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Section: Discussionmentioning

confidence: 98%

Sympathetic ophthalmia manifesting with conjunctival chemosis

et al. 2009

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“…Milder complications which respond to medical therapy include corneal erosions or dellen secondary to conjunctival chemosis in the early post-operative period [88] and delayed decompression-related reactivation, which has been described to occur in 1.3% of the cases a few weeks after surgery [89].…”

Section: Risks Of Decompression Surgerymentioning

confidence: 99%

Orbital Decompression in Thyroid Eye Disease

Boschi¹

2014

Orbital Surgery

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Thyroid-related orbitopathy (TRO) is part of a multisystemic disease mostly related to alterations of thyroid autoimmunity and characterized by a raised intraorbital pressure which causes disfigurement and functional deficits in the affected patient. Reduction of pathologically raised intraorbital pressure can be achieved by means of anti-inflammatory medical treatments or by decompression surgery which consist in enlarging the bony orbit and/or orbital fat removal. In the active phase of TRO, glucocorticoids are the first-line treatment of dysthyroid optic neuropathy; in the case of failed medical treatment, decompression surgery can be a valid alternative. Decompression surgery is the mainstay therapy for the treatment of stable typical disfiguring alterations and/or symptoms of TRO, although it has rarely been linked to reactivation of stable orbitopathy. The multifaceted nature of the disease, the different indications for decompression surgery, surgeon preferences and expertise, variations in orbital osteology, and patients' expectations and attitude toward intervention and possible complications imply the use of many different surgical techniques. Most of the techniques currently used seem to be effective in restoring optic nerve dysfunction, eye position, and/or congestive symptomatology and disfigurement; nevertheless, an unbiased analysis of the current literature in terms of effectiveness versus safety is extremely difficult due to the great heterogeneity of the patients included in the published studies and variations applied to surgical techniques. Fibrosis due to long-lasting orbital disease or possible consequence of retrobulbar irradiation administered in the early phase of TRO has been questioned as being a possible cause of poor distensibility and plasticity of the orbital soft tissues leading to diminished effectiveness of orbital expansion surgery. Recent investigations do not confirm this. Consecutive strabismus may complicate any decompression procedure, but its treatment is not particularly complicated.

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“…The decompression achieved is similar to that of the coronal approach, with less risk of postoperative diplopia [7,8]. However, the swinging eyelid approach occasionally causes chemosis (Figure 1a) [6] because this approach involves incisions in the lateral canthus and the conjunctiva in the lower eyelid, both of which contain lymphatic channels that drain the lower conjunctiva ( Figure 1b) [9]. Chemosis sometimes continues long after the operation, with resultant epiphora, ocular irritation, and visual disturbance [9,10].…”

Section: Introductionmentioning

confidence: 98%

“…The swinging eyelid approach is one of the procedures frequently used for deep lateral decompression [6]. This approach provides large surgical exposure, which includes the deep lateral orbital wall, the orbital floor, and part of the medial orbital wall.…”

Section: Introductionmentioning

confidence: 99%