“…Authors who have performed studies on cAVB complicating MI, we cite UJ Gang, Hymie H, Harpaz D, Nguyen [6][7][8][9] have worked on what appear to be patients with cAVB present during the early phase of an MI even before revascularization either by thrombolysis or by PR to judge the effectiveness of the two methods on the patient's prognosis. In the course of our research, we issued opinions, possible hypotheses, which could explain, if applicable, our patient: a) Either by stenosis of the Stent with spontaneous reperfusion by fibrinolysis which allows the lysis of the fibrinoerythro-platelet clot, and the maintenance of vascular permeability [10,11] (The in situ formation of a coronary thrombus can lead to the occlusion responsible for 'an MI, followed by spontaneous lysis of the thrombus, which may explain the discovery of normal angiography [12]); which is unlikely given that the patient received a 600 mg bolus of Clopidogrel, just prior to PR, to prevent activation of platelets by inhibiting adenosine Di-phosphate; b) Or by prolonged vasospasm of the anterior interventricular artery [11][12][13][14][15][16][17][18].…”