La r�action de Marchi: Aspects ultrastructuraux

Fuentes, C; Marty, Robert

doi:10.1007/bf00696569

Cited by 4 publications

(1 citation statement)

References 19 publications

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“…Comme antérieurement (Fuentes et Marty, 1975). les animaux ont été perfusés avec un mélange à parties égales de chlorate et de bichromate de potassium, pour mettre en évidence la réaction de Marchi.…”

Section: Microscopie Optiqueunclassified

Le nerf optique du chat

Fuentes¹,

Roch²,

Marty³

1979

Cells Tissues Organs

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54 healthy cats were studied. In almost all cases, two types of degenerative signs could be seen: in the primary optic pathways, but also in the posterior funiculi and the spinalcerebellar tracts, lesions of wide-diameter axons seen by their initial demyelination, accompanied by intense neurogJial reactions, and, on the other hand, a Wallerian degeneration of smaller axons situated in the axial part of the nerve. The percentage of these abnormalities must still be evaluated. Their eventual consequences and nature are discussed.

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Section: Microscopie Optiqueunclassified

Le nerf optique du chat

Fuentes¹,

Roch²,

Marty³

1979

Cells Tissues Organs

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Axonal regeneration in experimental allergic peripheral neuritis

Mervart

Kiernan

1978

Acta Neuropathol

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It has recently been suggested that severed axons fail to regenerate in the mammalian central nervous system as a result of an autoimmune reaction to myelin basic protein released into the circulation at the time of injury. Since the autoantigenic components of peripheral myelin are rapidly phagocytosed after axonal transection, it is claimed that a comparable immune response does not occur following injury to peripheral nerves, so the regenerative process is not hindered. If this contention is correct, it should be possible to inhibit the regeneration of peripheral axons by inoculating animals with suitable neuritogenic homogenates of peripheral nervous tissue. It has been shown that axonal regneration proceeds at the same rate in rats with experimental allergic neuritis as in healthy controls inoculated only with Freund's adjuvant. It is unlikely, therefore, that myelin basic proteins can stimulate the production of antibodies capable of inhibiting regenerative axonal growth.

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