“… 21 Due to the mutation in multiple intestinal neoplasia, Apc Min/+ mice spontaneously develop multiple adenomatous polyps in the small intestine and fewer polyps in the colon. 22 , 23 Therefore, Apc Min/+ mice remain a classic mouse model for studying human intestinal tumorigenesis. 24 , 25 , 26 , 27 The Apc Min/+ ; Mogat2 −/− mice model was established to investigate the effect of Mogat2 on spontaneous intestinal adenocarcinoma in mice with an APC mutation.…”