of left vertebral artery and reduced caliber of both the posterior cerebral arteries [ Figure 2]. Biochemistry was normal and procoagulant workup and also the workup for young stroke were negative. 2D echo was normal. Patient was put on antiplatelets and at three months follow-up his modified Rankin scale score was 2D echo.Cocaine, cannabis, amphetamines and marijuana are the common recreational drugs associated with ischemic and hemorrhagic strokes. The side-effects of acute cannabis intoxication include: (1) psychiatric -behavioral abnormalities and increased risk of schizophrenia; [1] (2) cardiovascular -tachycardia, supine hypertension, postural hypotension, myocardial infarction, paroxysmal atrial fibrillation and sudden death; [2] (3) cerebral -hemorrhagic and recurrent ischemic strokes. The mechanisms postulated in cannabis-related strokes are vasospasm, vasculopathy, postural hypotension with abnormal regulation of the cerebral blood flow and paroxysmal atrial fibrillation. [3] A predilection for infarcts in the posterior circulation as seen in our patient has been attributed to a poor autoregulatory mechanism in this vascular bed as compared to the anterior circulation. [4] Toxicological screening for cannabinoid metabolites should be done in young patients with strokes with no apparent vascular risk factors or evidence of dissection. This holds good even in our country with increasing drug abuse among the youth. 3. Cooles P, Michaud R. Stroke after heavy cannabis smoking. Postgrad Med J 1987;63:511. 4. Mateo I, Pinedo A, Gomez-Beldarrain M, Basterretxea JM, Garcia-Monco JC. Recurrent stroke associated with cannabis use.