2019
DOI: 10.1016/j.mehy.2019.01.009
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Lateral epicondylalgia: A primary nervous system disorder

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Cited by 10 publications
(8 citation statements)
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“…The conservative treatment included tablet Aceclofenac 100 mg if required and tablet Pantoprazole 40 mg if needed; rest from repetitive activities aggravating pain, deep friction massage, ice application, stretching exercises, grip strengthening and counterforce bracing. [ 6 ] Conservative management was continued throughout the study period irrespective of the management response.…”
Section: Methodsmentioning
confidence: 99%
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“…The conservative treatment included tablet Aceclofenac 100 mg if required and tablet Pantoprazole 40 mg if needed; rest from repetitive activities aggravating pain, deep friction massage, ice application, stretching exercises, grip strengthening and counterforce bracing. [ 6 ] Conservative management was continued throughout the study period irrespective of the management response.…”
Section: Methodsmentioning
confidence: 99%
“…A theoretical framework noting that the nervous system is likely one component in causing pain in LE emphasized that the excessive expansion of neuronal action and the loss of homeostatic regulation by RN-derived nociceptors contribute to axonal growth free sensitive spots. [ 6 ] The present study's focus was not on radial nerve (RN) compression or entrapment neuropathy as the primary driver of the pathophysiology but the RN's role in peripheral sensitization.…”
Section: Introductionmentioning
confidence: 99%
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“…Most of the time, this injury is considered as a tendinopathy because lateral epicondylalgia often presents with a degenerative process that affects the tendon structure, although motor system impairments and changes in pain perception have also been observed [ 6 , 7 ]. Changes can be seen at the histological level with disorganization in the arrangement of collagen fibers and local hypervascularization, proliferation of fibroblasts, and tissue granulation [ 8 , 9 , 10 ]. Specifically, in LE, there are changes in the tendon of the extensor carpi radialis brevis muscle [ 7 , 9 , 11 ], and usually, the pain is intensified by resisted muscular contraction of the extensor carpi radialis brevis [ 3 , 7 ].…”
Section: Introductionmentioning
confidence: 99%
“…Changes can be seen at the histological level with disorganization in the arrangement of collagen fibers and local hypervascularization, proliferation of fibroblasts, and tissue granulation [ 8 , 9 , 10 ]. Specifically, in LE, there are changes in the tendon of the extensor carpi radialis brevis muscle [ 7 , 9 , 11 ], and usually, the pain is intensified by resisted muscular contraction of the extensor carpi radialis brevis [ 3 , 7 ]. On ultrasound examination, it is possible to observe thickening of the tendon, areas of hypogenicity, and neovascularization [ 4 ], as well as the existence of angiofibroblastic hyperplasia [ 7 ].…”
Section: Introductionmentioning
confidence: 99%