Lateral medullary syndrome

Sundarachary, N V; Amalakanti, Sridhar

doi:10.15380/2277-5706.jcsr.14.074

Cited by 2 publications

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“…The risk factors for LMS occurrence are the same as for other types of ischemic stroke: hypertension, smoking, diabetes, and atrial fibrillation [ 4 ]. The common pathological mechanisms for lateral medullary infarction are atherosclerosis, arterial dissection, cardiogenic emboli, and small vessel disease in decreasing frequency of occurrence [ 5 ] [ 6 ].…”

Section: Introductionmentioning

confidence: 99%

Sinus arrest following acute lateral medullary infarction

Alloush

Sami

et al. 2022

Neurol Sci

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Lateral medullary syndrome (LMS) is an ischemic stroke of the medulla oblongata that involves the territory of the posterior inferior cerebellar artery. LMS is often missed as the cause of autonomic dysregulation in patients with recent brain stem stroke. Due to the location of the nucleus tractus solitarius (NTS), the dorsal vagal nucleus, and the nucleus ambiguous in the lateral medulla oblongata, patients with LMS occasionally have autonomic dysregulation–associated clinical manifestations. We report a case of LMS-associated autonomic dysregulation. The case presented by recurrent syncope, requiring permanent pacemaker placement. This case shows the importance of recognizing LMS as a potential cause of life-threatening arrhythmias, heart block, and symptomatic bradycardia. Extended cardiac monitoring should be considered for patients with medullary strokes.

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Section: Introductionmentioning

confidence: 99%

Sinus arrest following acute lateral medullary infarction

Alloush

Sami

et al. 2022

Neurol Sci

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Rehabilitation and Physiotherapy Action Strategy for an Acute Case of Lateral Medullary Syndrome: A Case Report

Kariya,

Singh,

Sheikh

2024

Cureus

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Lateral medullary syndrome (LMS) may result from a failure in either the vertebral artery or the posterior inferior cerebellar artery. Stroke is the most common cause of LMS. To achieve bronchial hygiene and improve the patient's condition, chest physiotherapy was initiated due to his confined condition during the acute stage. As a result, a four-week physiotherapy program was established and administered twice daily to the patient, with noticeable results. A physical examination revealed that the patient exhibited tachycardia, dyspnea, intracranial nerve palsies on the left side, thermoanesthesia on the right, horizontal nystagmus, and ptosis and miosis in the left eye associated with Horner syndrome. After receiving appropriate conservative care, the patient was discharged from the hospital with the fewest possible disabilities.

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Lateral medullary syndrome

Cited by 2 publications

References 0 publications

Sinus arrest following acute lateral medullary infarction

Sinus arrest following acute lateral medullary infarction

Rehabilitation and Physiotherapy Action Strategy for an Acute Case of Lateral Medullary Syndrome: A Case Report

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