1965
DOI: 10.1016/0002-8703(65)90186-9
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Left axis deviation induced experimentally in a primate heart

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1968
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Cited by 78 publications
(14 citation statements)
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“…Watt and Pruitt (17), using intact dog hearts, were able to produce some epicardial delay with rather large septal lacerations in the distribution of the major divisions but could not produce significant frontal axis shifts. Watt et al were able to produce axis shifts in a primate heart (35), and attributed their results to species differences in the conducting system.…”
Section: Myerburg Nillson Gelbandmentioning
confidence: 99%
“…Watt and Pruitt (17), using intact dog hearts, were able to produce some epicardial delay with rather large septal lacerations in the distribution of the major divisions but could not produce significant frontal axis shifts. Watt et al were able to produce axis shifts in a primate heart (35), and attributed their results to species differences in the conducting system.…”
Section: Myerburg Nillson Gelbandmentioning
confidence: 99%
“…The explanation for this in IMI is probably contained in the classic concept of unopposed and enhanced anterior and superior forces due to the loss of muscle posteroinferiorly. Because the early transseptal forces may be maintained intact, the departure from normal does not begin in this group until well within or after the normal Q time zone.12 Experimentally produced LAFB in animals has been found to shift the mean QRS axis superiorly23 24 and to delay the activation time of the epicardium of the lateral basal surface of the left ventricle.25 The normal net direction of spread of depolarization, therefore, is presumably altered in LAFB, permitting abnormal spread of depolarization anteriorly and superiorly from the region already activated through the left posterior fascicle. Thus, this abnormal, superiorly directed positivity in LAFB arises from slight initial delay, sustained dysynchrony, and abnormality of direction of activation of the territory supplied by the left anterior fascicle.…”
Section: Discussionmentioning
confidence: 95%
“…These pathophysiological considerations suggest, therefore, that a selective histological investigation of the conducting system would be indicated rather than a search for pathological 'foci' somewhere in the heart. Indeed, lesions of the conducting system are not only credited with enhancing cardiac desynchronization in general, but the lesions of the bundle-branches, the left in particular, are held to have a precise bearing on shifts of QRS axis (Watt, Murao, and Pruitt, 1965;Rosenbaum, 1970).…”
Section: Discussionmentioning
confidence: 99%