1977
DOI: 10.1016/s0002-9149(77)80006-4
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Left ventricular function after cigarette smoking by chronic smokers: Comparison of normal subjects and patients with coronary artery disease

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Cited by 26 publications
(13 citation statements)
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“…We have shown that smoking a nonnicotine, nontobacco cigarette does not alter systolic time interval measurements, and, recently, Jain et al 22 reported that the systolic time interval-derived ratio of PEP/LVET increased in chronic smokers with coronary artery disease but decreased in healthy volunteers after smoking two nicotine tobacco cigarettes, confirming our preliminary report.15 Studies by Aronow et al in tion of several tobacco cigarettes showed a decrease in stroke index and EF documented by left ventriculography.23 They attributed this negative inotropic effect to rising levels of carboxyhemoglobin impairing oxygen delivery to an already compromised myocardium.…”
Section: Discussionsupporting
confidence: 52%
“…We have shown that smoking a nonnicotine, nontobacco cigarette does not alter systolic time interval measurements, and, recently, Jain et al 22 reported that the systolic time interval-derived ratio of PEP/LVET increased in chronic smokers with coronary artery disease but decreased in healthy volunteers after smoking two nicotine tobacco cigarettes, confirming our preliminary report.15 Studies by Aronow et al in tion of several tobacco cigarettes showed a decrease in stroke index and EF documented by left ventriculography.23 They attributed this negative inotropic effect to rising levels of carboxyhemoglobin impairing oxygen delivery to an already compromised myocardium.…”
Section: Discussionsupporting
confidence: 52%
“…In addition, the percentage of RA‐NPV triggers was higher in smokers than in nonsmokers and ablation outcome was worse in smokers with RA‐NPV. We hypothesized that smoking causes poor ablation outcome in smokers with RA‐NPV via mechanisms mentioned above …”
Section: Discussionmentioning
confidence: 99%
“…10 In the presence of coronary-artery stenosis, the coronary flow can not increase and ischemia may develop, with resultant angina pectoris or myocardial dysfunction. 11,12 Recently, Barua RS, et al demonstrated that smoking could cause dysfunction of the nitric oxide biosynthesis in endothelium cells by oxdative stress 9 and that mechanism may further impair the endothelium-dependent coronary artery dilatation capability and reduce the myocardial oxygen supply.…”
Section: Cardiopulmonary Effect Of Smokingmentioning
confidence: 99%
“…[10][11][12][13][14] Cigarette consumption is directly related to increased rates of sudden cardiac death (SCD), mainly through its direct toxic effects. Recently, the impact of the smoking status on SCD was studied in patients with established coronary artery 15 In that study, current cigarette smoking was a powerful independent predictor of the SCD risk and those that quit smoking experienced a significant reduction in the SCD risk.…”
Section: Smoking and Cardiac Arrhythmiasmentioning
confidence: 99%