Lesions found at foot trimming of dairy goats: Baseline data for comparing lesions and locomotion scoring

Chesterton, R.N.; Chesterton, S.; Laven, Richard

doi:10.1016/j.tvjl.2022.105927

Cited by 5 publications

(2 citation statements)

References 17 publications

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“…Given the potential economic impact, lameness prevention, and control could contribute to farm efficiency, sustainability, and productivity by improving goat welfare. Two New Zealand studies have investigated lameness [ 2 , 3 ]. The first study followed a small cohort of goats during their first two years of life [ 2 ].…”

Section: Introductionmentioning

confidence: 99%

“…Deeming et al [ 2 ] reported a small percentage (0 to 8.9%) of lame goats within the group across four locomotion-scoring events after parturition. The second study recorded lameness prevalence between 6.7 and 25.5% in four dairy goat herds [ 3 ]. It is unknown what causes lameness in dairy goat herds and whether claw disorders are the predominant reason for lameness.…”

Section: Introductionmentioning

confidence: 99%

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Estimates of Genetic Parameters for Milk, the Occurrence of and Susceptibility to Clinical Lameness and Claw Disorders in Dairy Goats

Jaques

Turner

Vallée³

et al. 2023

Animals

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The New Zealand goat industry accesses niche markets for high-value products, mainly formula for infants and young children. This study aimed to estimate the genetic parameters of occurrence and susceptibility of clinical lameness and selected claw disorders and establish their genetic associations with milk production traits. Information on pedigree, lameness, claw disorders, and milk production was collected on three farms between June 2019 and July 2020. The dataset contained 1637 does from 174 sires and 1231 dams. Estimates of genetic and residual (co)variances, heritabilities, and genetic and phenotypic correlations were obtained with uni- and bi-variate animal models. The models included the fixed effects of farm and parity, deviation from the median kidding date as a covariate, and the random effects of animal and residual error. The heritability (h2) estimates for lameness occurrence and susceptibility were 0.07 and 0.13, respectively. The h2 estimates for claw disorder susceptibilities ranged from 0.02 to 0.23. The genotypic correlations ranged from weak to very strong between lameness and milk production traits (−0.94 to 0.84) and weak to moderate (0.23 to 0.84) between claw disorder and milk production traits.

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Section: Introductionmentioning

confidence: 99%

Section: Introductionmentioning

confidence: 99%