Linear lichen planus pigmentosus: A rare entity with an illusory presentation

Das, Anupam; Mishra, Vivek; Podder, Indrashis; Kumar, Piyush; Das, Dipti; Das, Nilay Kanti

doi:10.4103/2349-5847.147048

Cited by 15 publications

(28 citation statements)

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“…7 The majority of LPP lesions are usually distributed in areas exposed to the sun; trunk lesions, such as those observed in the present case, are rarely observed. 8 Both topical and oral treatments for LPP have been used with varying results, including topical steroids, keratolytics, tacrolimus, griseofulvin, prednisone, etretinate and chloroquine. 3,4 dermatosis with hyperpigmentation and drug-related melanoses.…”

Section: Commentmentioning

confidence: 99%

Lichen Planus Pigmentosus with Blaschkoid Distribution

Alonso-Corral¹,

Garrido‐Colmenero²,

Almódovar-Real³

et al. 2016

SQUMJ

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interesting medical image A 57-year-old woman presented at a dermatology unit in Granada, Spain, in 2015 with a two-month history of slightly pruritic brown-coloured macular lesions on the trunk and right arm. She was not taking any medications or using cosmetics and had not been exposed to the sun before the onset of the rash. On examination, clearly defined hyperpigmented brown macules were seen on the right submammary region, right hemithorax, right abdomen and right buttock [ Figure 1] with an S-shaped appearance, compatible with the pattern of Blaschko lines. Tests for hepatitis B and hepatitis C were negative. A histological examination showed rich lymphocytic infiltrate and melanin incontinence with slight epidermal atrophy and lichenoid reaction with hypergranulosis [ Figure 2]. Considering these features, a diagnosis of lichen planus pigmentosus (LPP) with Blaschkoid distribution was proposed. The patient was prescribed topical clobetasol propionate for four weeks. The lesions responded well to the treatment, with significant pigment reduction and improvement of the associated pruritus. CommentVarious triggering factors, ranging from viral infections and vaccinations to trauma, have been implicated in the aetiology of lichen planus.1 A rare variant of lichen planus, LPP is distinguished by hyperpigmented macules or papules. The histological characteristics of LPP include epidermal atrophy, vacuolar degeneration of the basal cell layer and rich dermal lichenoid infiltrate.2 Kanwar et al. suggested that LPP may represent a lichenoid reaction to an unknown agent or stimuli, with regards to the histological concordance between LPP and lichen planus.3 The differential diagnosis of LPP includes conditions such as lichen striatus, erythema dyschromicum perstans, incontinentia pigmenti, linear and whorled nevoid hypermelanosis, occupational Sultan Qaboos University Med J, August 2016, Vol. 16, Iss. 3, pp. e383-384,

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Section: Commentmentioning

confidence: 99%

Lichen Planus Pigmentosus with Blaschkoid Distribution

Alonso-Corral¹,

Garrido‐Colmenero²,

Almódovar-Real³

et al. 2016

SQUMJ

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“…Face and neck are the frequent initial sites of onset followed by involvement of other body sites including body folds (LPP inversus). 15,16 Its exact etiology is poorly understood but UV light, viral infections, and topical mustard oil and amla oil have been often implicated. LPP in 10 (3.3%) patients was not associated with any systemic disease and HBsAg and Anti-HCV serology was negative (Fig.…”

Section: Resultsmentioning

confidence: 99%

Clinical profile of 300 men with facial hypermelanosis

Gupta¹,

Mahajan²

2017

J Dermatol Case Rep

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Background: Facial hypermelanosis is a significant cause of cosmetic disfigurement, social embarrassment and psychological morbidity affecting quality of life.Objective: To study clinicoepidemlogic patterns of facial hypermelanoses among men. Material and Methods:Medical records of all adult males presenting with facial hypermelanoses were analyzed for this retrospective cross sectional study for demographic details, duration, cosmetic usage, sun exposure, drug intake, infections, systemic or cutaneous diseases, and family history of hypermelanotic dermatosis. Laboratory investigations and skin biopsy were performed when deemed necessary.Results: These were 300 Indian men aged 18 to 74 (mean 37.35) years with 121 (40.3%) individuals aged 31-50 years. Various patterns of melasma in 230 (76.7%) patients were the major cause of facial hypermelanosis. Periorbital hypermelanosis was observed in 32 (10.7%), freckles and lentigens in 26 (8.7%), acanthosis nigricans in 12 (4%) and lichen planus pigmentosus in 10 (3.3%), pigmented cosmetic contact dermatitis in 7, and nevus of Ota in 6 persons. The 71 (30.8%) patients with melasma had a history of frequent sun exposure, 9 (3.9%) patients had systemic comorbidities. Family history of periorbital melanosis was present in 7 (21.8%), personal or family history of atopy in 5 (15.6%) patients. Acanthosis nigricans was associated with obesity in 9 (75%) of patients and with diabetes mellitus in 4 (33.3%) cases.Conclusions: Melasma, periorbital hypermelanosis, acanthosis nigricans and lichen planus pigmentosus remain the predominant causes for facial hypermelanosis in men.

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“…Linear LPP is an extremely rare clinical variant of LP. [1][2][3][4][5][6][7][8][9] Unlike LP, LPP often spares the scalp, mucous membranes, and nails, 7,9 presenting in various forms-diffuse, linear, zosteriform, segmental, reticular, perifollicular. 6,9 It is idiopathic in nature, 1,2,4,6,9 although like LP, hepatitis C virus has been reported to be a possible etiology.…”

Section: Discussionmentioning

confidence: 99%

“…[1][2][3][4][5][6][7][8][9] Unlike LP, LPP often spares the scalp, mucous membranes, and nails, 7,9 presenting in various forms-diffuse, linear, zosteriform, segmental, reticular, perifollicular. 6,9 It is idiopathic in nature, 1,2,4,6,9 although like LP, hepatitis C virus has been reported to be a possible etiology. 7,9,12 It may be associated with exposure to metal implants (e.g., gold), 1,2 medications (e.g., ibuprofen), 1,5,6 trauma (Koebner's phenomenon), 3,4 sunlight, 3,4,6,7 mustard and amla oils, henna, fragrances, and hair dyes.…”

Section: Discussionmentioning

confidence: 99%

“…This idiopathic and inflammatory skin condition commonly affects dark-skinned individuals along sun-exposed areas (face: along the facial lines of Blaschko, neck, limbs) and flexural folds (axilla, inguinal, submammary). [1][2][3][4][5][6][7][8][9] Periorbital linear LPP is an even rarer clinical entity with only a few recently published reports of this benign dermatosis affecting the periorbital region. 10,11 There has also been a published case report of bilateral periorbital lichen nitidus; this is, however, a separate clinical entity from periorbital linear LPP.…”

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