Lipomembranous changes in chronic panniculitis

Alegre, V.; Winkelmann, R. K.; Aliaga, Adolfo

doi:10.1016/s0190-9622(88)70149-8

Cited by 65 publications

(49 citation statements)

References 6 publications

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“…In 1977, Akai et al reported 6 cases of membranous lipodystrophy with concomitant intra-osseous membranocystic lesions and neuropsychiatric disturbances [1]. It was almost a decade later that MFN of extra-osseous systemic tissue was reported [2,3,8,12,13]. The reported systemic adipose tissue sites in which MFN has been described to date include the breast, subcutis, chest wall and scrotum, in the clinical settings of diabetes mellitus, panniculitis, granulomatous inflammation and systemic lupus erythematosus [16].…”

Section: Discussionmentioning

confidence: 99%

Membranous fat necrosis in appendices epiploicae

Ramdial

Singh

1998

Virchows Archiv

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Membranous fat necrosis (MFN) is a degenerative process involving mature systemic adipose tissue. It is characterised by the presence of membranocystic foci surrounded by a lipophagic fibro-inflammatory reaction typical of fat necrosis. Membranocystic foci are cysts lined by an eosinophilic membrane with pseudopapillary infoldings having the histochemical staining profile of ceroid. Although MFN is described in an increasing number of adipose tissue sites, it has not been described as a distinct entity in appendices epiploicae (AE). Macroscopically, MFN in AE mimics nodal tuberculosis or metastatic tumour with necrosis and cystic change. Ischaemia, which can be secondary to physiological or pathologic processes, is crucial in the pathogenesis of MFN in AE. Heightened awareness of MFN as a distinct entity in AE is essential for accurate diagnosis and establishment of the pathogenesis of this enigmatic pathological process.

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Section: Discussionmentioning

confidence: 99%

Membranous fat necrosis in appendices epiploicae

Ramdial

Singh

1998

Virchows Archiv

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“…To date, LFN has been found in: leg ischaemic necrosis secondary to chronic arterial obstruction; 4 patients with venous insufficiency, thromboangiitis obliterans and collagenopathies (lupus erythematosus, scleroderma and lipogranulomas); 5 lipomas; 6 diabetic patients; erythema nodosum; and patients with stasis dermatitis and lipogranulomas 7 . Approximately 16% of patients with LFN do not present any associated, underlying disease 8 , 9 .…”

Section: Introductionmentioning

confidence: 99%

Lipomembranous fat necrosis in three cases of testicular torsion

2001

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“…It is generally accepted that this change lacks specificity and may be found in many longstanding lesions of many variants of panniculitis such as EN, lupus panniculitis, Bazin disease, necrobiosis lipoidica, subcutaneous sarcoidosis, and traumatic panniculitis. The mechanism for the formation of pseudomembranes in the adipose tissue remains unknown . Fat infarction in LDS could be secondary to local stasis changes in the subcutaneous microvasculature, which may have an effect on transcapillary oxygen exchange.…”

Section: Discussionmentioning

confidence: 99%