Living donor liver transplantation with extensive caval thrombectomy for acute-on-chronic Budd-Chiari syndrome

Soyama, Akihiko; Eguchi, Susumu; Yanaga, Katsuhiko; Takatsuki, Mitsuhisa; Hidaka, Masaaki; Kanematsu, Takashi

doi:10.1007/s00595-010-4376-z

Cited by 15 publications

(8 citation statements)

References 10 publications

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“…Various treatments, including transjugular intrahepatic portosystemic shunt (TIPS), surgical shunts, and liver transplantation have been described as potential treatment options for BCS (3)(4)(5)(6). However, there are only a few studies on accessory hepatic vein (AHV) recanalization for treatment of BCS.…”

mentioning

confidence: 99%

Accessory hepatic vein recanalization for treatment of Budd-Chiari syndrome due to long-segment obstruction of the hepatic vein: initial clinical experience

Xu²,

Zhang³

et al. 2015

Diagn Interv Radiol

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mentioning

confidence: 99%

Accessory hepatic vein recanalization for treatment of Budd-Chiari syndrome due to long-segment obstruction of the hepatic vein: initial clinical experience

Xu²,

Zhang³

et al. 2015

Diagn Interv Radiol

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“…The patient's Child-Pugh and Model for End-stage Liver Disease scores were 11 and 23, respectively (www.mayoclinic.org/meld/mayomodel6.html). Preoperative laboratory data showed a platelet count of 185.000 mm 3 , hemoglobin level of 13,2 g/dL, activated partial thromboplastin time of 34.7, and international normalized ratio of 1.14 (0.9-1.4). Physical examination was significant for a massively distended abdomen secondary to ascites.…”

Section: Case Reportmentioning

confidence: 99%

“…Budd Chiari syndrome (BCS) is a rare disorder resulting from hepatic venous outflow tract with obstruction anywhere from the small hepatic veins to the suprahepatic inferior vena cava [3]. In these patients, hemodynamic instability can be dramatically diminished by inferior vena cava cross-clamping during OLT [4].…”

Section: Introductionmentioning

confidence: 99%

Management of Budd Chiari Syndrome by liver transplantation: A case report

Aydoğan¹,

Kayaalp²,

Bıçakcıoğlu³

et al. 2013

CMJ

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Budd-Chiari syndrome (obstruction of venous drainage of the liver) may be idiopathic, due to thrombosis of the hepatic veins in thrombogenic states or be due to extrinsic compression. It is rare, but life-threatening, if left untreated. For patients with end-stage liver disease, liver transplantation should be considered. Orthotopic liver transplantation (OLT) is associated with the potential for massive blood loss, necessitating rapid infusion of large quantities of blood products. In these patients, hemodynamic instability can be dramatically diminished by inferior vena cava cross-clamping during OLT. There are few reports about living donor transplantations in BuddChiari case. We present a case of 27-year-old man, with a diagnosis of end-stage liver disease secondary to Budd-Chiari syndrome and underwent living OLT. Anaesthesia management, and follow-up of patients affected by this condition represent a challenge for anesthesiologists. The aim of this report is to discuss the potential options in the intraoperative management of patients with Budd-Chiari syndrome, who will undergo living OLT.

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“…[1][2][3] There are a few reports of surgical thrombectomy of the vena cava during orthotopic liver transplantation, 1,3 but there have been none without a shunt or venovenous bypass.…”

mentioning

confidence: 99%

“…Soyama et al 3 recently performed successful living donor liver transplantation with extensive thrombectomy in a patient with acute-on-chronic Budd-Chiari syndrome and a totally thrombosed retrohepatic IVC. In that procedure, a Fogarty catheter was used to remove the thrombus, the IVC was opened longitudinally and IVC venoplasty was performed.…”

mentioning

confidence: 99%

Vena cava thrombectomy during orthotopic liver transplantation without a venovenous bypass

et al. 2012

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A series of novel vanadium(III) complexes bearing heteroatom‐containing group‐substituted salicylaldiminato ligands [RNCH(ArO)]VCl2(THF)2 (Ar = C6H4, R = C3H2NS, 2a; C7H4NS, 2c; C7H5N2, 2d; Ar = C6H2tBu2 (2,4), R = C3H2NS, 2b) have been synthesized and characterized. Structure of complex 2c was further confirmed by X‐ray crystallographic analysis. The complexes were investigated as the catalysts for ethylene polymerization in the presence of Et2AlCl. Complexes 2a–d exhibited high catalytic activities (up to 22.8 kg polyethylene/mmolV h bar), and affording polymer with unimodal molecular weight distributions at 25–70 °C in the first 5‐min polymerization, whereas produced bimodal molecular weight distribution polymers at 70 °C when polymerization time prolonged to 30 min. The catalyst structure plays an important role in controlling the molecular weight and molecular weight distribution of the resultant polymers produced in 30 min polymerization. In addition, ethylene/hexene copolymerizations with catalysts 2a–d were also explored in the presence of Et2AlCl, which leads to the high molecular weight and unimodal distributions copolymers with high comonomer incorporation. Catalytic activity, comonomer incorporation, and polymer molecular weight can be controlled over a wide range by the variation of catalyst structure and the reaction parameters, such as comonomer feed concentration, polymerization time, and polymerization reaction temperature. © 2009 Wiley Periodicals, Inc. J Polym Sci Part A: Polym Chem 47: 3573–3582, 2009

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Living donor liver transplantation with extensive caval thrombectomy for acute-on-chronic Budd-Chiari syndrome

Cited by 15 publications

References 10 publications

Accessory hepatic vein recanalization for treatment of Budd-Chiari syndrome due to long-segment obstruction of the hepatic vein: initial clinical experience

Accessory hepatic vein recanalization for treatment of Budd-Chiari syndrome due to long-segment obstruction of the hepatic vein: initial clinical experience

Management of Budd Chiari Syndrome by liver transplantation: A case report

Vena cava thrombectomy during orthotopic liver transplantation without a venovenous bypass

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