Localised alveolar-septal amyloidosis with hypersensitivity pneumonitis

Orriols, Ramón; Aliaga, J.L.; Morell, Ferrán; Rodrigo, María-José; García, Fernando; Royo, Luis José

doi:10.1016/0140-6736(92)91595-y

Cited by 7 publications

(2 citation statements)

References 9 publications

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“…This suggests that the distinction proposed by some authors between arteriolar amyloidosis and alveolar septal amyloidosis is not clear [4,5]. Progressive exertional dyspnea is the most common symptom, and its severity depends on the degree of infiltration of the alveolarcapillary gas exchange zone, and thus pulmonary function tests usually show a pattern of restrictive lung disease [2,5,6]. Chest radiograph shows a non-specific diffuse interstitial reticulonodular infiltration suggestive of interstitial lung disease.…”

Section: Discussionmentioning

confidence: 94%

CT features of primary systemic pulmonary amyloidosis mimicking pulmonary sarcoidosis

et al. 2012

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Section: Discussionmentioning

confidence: 94%

CT features of primary systemic pulmonary amyloidosis mimicking pulmonary sarcoidosis

et al. 2012

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“…Interstitial fibrosis was also observed in two patients, and granulomata in another two. Amyloid deposits were observed in one patient described previously [16].…”

Section: Cross-sectional Studymentioning

confidence: 85%

High prevalence of mollusc shell hypersensitivity pneumonitis in nacre factory workers

Orriols¹,

Aliaga²,

Antó³

et al. 1997

Eur Respir J

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Following the discovery of hypersensitivity pneumonitis caused by the inhalation of mollusc shell dust in two workers from a nacre-button factory, the health status of 26 workers employed in sawing mollusc shells was investigated. The evaluation included the administration of two questionnaires and radiological, functional and immunological assessments of all workers at the outset and 1 year later, when hygienic and therapeutic measures had been taken. Six workers, in whom specific inhalation challenge test was positive, were diagnosed with mollusc shell hypersensitivity pneumonitis, thus yielding a prevalence of 23%. Evidence of diffuse lung disease and systemic symptoms was found in these patients. Nonspecific bronchial hyperreactivity was also found more frequently in patients with mollusc shell hypersensitivity pneumonitis. Specific immunoglobulin G (IgG) level and specific skin testing failed to differentiate patients with mollusc shell hypersensitivity pneumonitis from other exposed workers; whereas, nonspecific skin testing, which was impaired in the patients, did differentiate. Bronchoalveolar lavage and transbronchial biopsy performed in patients with mollusc shell hypersensitivity pneumonitis were consistent with the disease. Removal from an environment containing mollusc shell dust was followed by regression of clinical, radiological and functional changes. The clinical picture of the 20 workers who did not present mollusc shell hypersensitivity pneumonitis remained unchanged, but functional decline was observed despite improvement in the environmental conditions of the factory. This report describes the first series of patients with mollusc shell hypersensitivity pneumonitis studied, and underlines the importance of careful follow-up of workers occupationally-exposed to mollusc shell dust.

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Hypersensitivity Lung Diseases

Seaton¹

2000

Crofton and Douglas's Respiratory Diseases

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Localised alveolar-septal amyloidosis with hypersensitivity pneumonitis

Cited by 7 publications

References 9 publications

CT features of primary systemic pulmonary amyloidosis mimicking pulmonary sarcoidosis

CT features of primary systemic pulmonary amyloidosis mimicking pulmonary sarcoidosis

High prevalence of mollusc shell hypersensitivity pneumonitis in nacre factory workers

Hypersensitivity Lung Diseases

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