Location of theSas-1 locus on mouse chromosome 1

Rosenstreich, David L.; Groves, Michael G.; Hoffman, Harold A.; Taylor, Benjamin A.

doi:10.1007/bf01844021

Cited by 18 publications

(4 citation statements)

References 14 publications

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“…Since previous studies indicated 17.5 k 3.9 for Pep-3 to LyM-1 (Sato et al,198 l), the probable gene order may be Pep-3-Sas-1-LyM-I-MIS. This inference is consistent with the report that Sas-I is located distal to Pep-3 (formerly called Dip-I) at a map distance of 3.0 5 1.5 (Rosenstreich et al, 1978).…”

Section: R E S U L T S a N D Discussionsupporting

confidence: 92%

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LOCATION OF LyM‐1 LOCUS ON MUS MUSCULUS CHROMOSOME 1

Satô

Natsuume‐Sakai

Katagiri

et al. 1981

Int J Immunogenetics

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Summary Following segregation of the alleles at the Sas‐1, LyM‐1 and M1s in the (AKR/J x C3H/HeJ)F1 x C3H/HeJ backcross progeny, we found the locus responsible for the lymphocyte alloantigen LyM‐1 to be closely linked to Sas‐1 and M1s. The map distances are estimated to be 12.5 ± 3.9 for Sas‐1 to LyM‐1, 6.9 ± 3.0 for LyM‐1 to M1s and 19.4 ± 4.7 for Sas‐1 to M1s. These data clearly indicate that the gene order is Sas‐1‐LyM‐1‐M1s on Mus musculus chromosome 1.

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Section: R E S U L T S a N D Discussionsupporting

confidence: 92%

“…The Sas-I and M I S loci were assigned to chromosome 1 (Rosenstreich et al, 1978;Festenstein et al, 1977) and the linkage of LyM-I to M l s has been established (Sato et al, 1981). A three-point genetic test was carried out to determine the position of LyM-1 with respect to Sas-I and MIS.…”

Section: R E S U L T S a N D Discussionmentioning

confidence: 99%

LOCATION OF LyM‐1 LOCUS ON MUS MUSCULUS CHROMOSOME 1

Satô

Natsuume‐Sakai

Katagiri

et al. 1981

Int J Immunogenetics

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“…En-1 alleles were typed with the mp probe as described in the text. The SDP for Pep-3 was determined by Wilson et al {1978} and for Sas-1 by Rosenstreich et al (1978). b The letters B, H, and C represent the alleles inherited from progenitor strains C57BL/6J, C3H/HeJ, and BALB/c, respectively.…”

Section: Discussionmentioning

confidence: 99%

En-1 and En-2, two mouse genes with sequence homology to the Drosophila engrailed gene: expression during embryogenesis.

Joyner¹,

Martin²

1987

Genes Dev.

320

157

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Development in multicellular organisms can be viewed as a two-step process. First, a general body plan is established, and subsequently the various components of the basic body plan diversify and specialize. It has not been obvious how to identify or isolate the genes responsible for the control of these processes in vertebrate species. However, the existence of mutations that perturb these processes in Drosophila melanogaster has led to the identification of three groups of genes that control development in that organism: The maternal effect and segmentation genes function to establish a body plan composed of repeating segmental units which are subdivided tPresent address: Division of Molecular and Developmental Biology, Mt.

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“…The' presumptive linkage of resistance with a chromosome 1 marker is of interest because resistance to two other intracellular pathogens, Salmonella typhimurium (20) and Leishmania donovani (7), is also linked to chromosome 1 markers. The locus (Lsh) controlling susceptibility to visceral leishmaniasis has been mapped between the centromere and the marker isocitrate dehydrogenase-1, which is proximal to Sas-1 (7,23). The locus controlling resistance to S. typhimurium is closely linked to but distinct from the Lsh locus (18,26).…”

Section: Discussionmentioning

confidence: 99%

Genetics of natural resistance to Sendai virus infection in mice

Brownstein¹

1983

Infect Immun

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The genetics of resistance to a naturally occurring respiratory infection caused by Sendai virus was examined in F1, F2, and backcross progeny of resistant C57BL/6J and susceptible DBA/2J mice and in 25 recombinant inbred strains. An intranasal inoculum of 0.1 50% tissue culture infective dose (low dose) of Sendai virus caused 0% mortality in C57BL/6J and F1 mice and 73% mortality in DBA/2J mice. An inoculum of 1.0 50% tissue culture infective dose (high dose) caused 3, 0, and 89% mortality in C57BL/6J, F1, and DBAI2J mice, respectively. Low-dose infection caused 36% mortality in F1 x DBA/2J hybrids and 0% mortality in F2 hybrids. High-dose infection caused 29 and 32% mortality in F1 x DBA/2J and F2 hybrids, respectively. Resistance was not linked to H-2 haplotype, coat color, or sex. High-dose infection caused deaths in 12 recombinant inbred strains, and the strain distribution pattern was concordant with that of a chromosome 1 marker, Sas-1, in 20 of 25 strains (P < 0.01). Resistance therefore behaved as a simple on August 4, 2020 by guest

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Location of theSas-1 locus on mouse chromosome 1

Cited by 18 publications

References 14 publications

LOCATION OF LyM‐1 LOCUS ON MUS MUSCULUS CHROMOSOME 1

LOCATION OF LyM‐1 LOCUS ON MUS MUSCULUS CHROMOSOME 1

En-1 and En-2, two mouse genes with sequence homology to the Drosophila engrailed gene: expression during embryogenesis.

Genetics of natural resistance to Sendai virus infection in mice

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