2001
DOI: 10.1016/s0002-9149(01)01577-6
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Long-term (≥8 years) outcome after palmaz-schatz stent implantation

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Cited by 51 publications
(39 citation statements)
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“…Long-term outcome of clinical follow-up in the present study confirmed the observation of Choussat et al, 10 who reported the rate of revascularization of stented sites was Ͻ10% beyond 1 year for up to 10 years, and late revascularization was performed predominantly to treat progressive disease at nontarget sites. Therefore, the clinical benefits of coronary stenting in terms of reduction in the rate of TL revascularization seemed to be sustained at 7 to 11 years of follow-up.…”
Section: Discussionsupporting
confidence: 90%
See 1 more Smart Citation
“…Long-term outcome of clinical follow-up in the present study confirmed the observation of Choussat et al, 10 who reported the rate of revascularization of stented sites was Ͻ10% beyond 1 year for up to 10 years, and late revascularization was performed predominantly to treat progressive disease at nontarget sites. Therefore, the clinical benefits of coronary stenting in terms of reduction in the rate of TL revascularization seemed to be sustained at 7 to 11 years of follow-up.…”
Section: Discussionsupporting
confidence: 90%
“…Medium-term follow-up studies at 3 to 5 years revealed a paucity of late clinical stent-related problems 4 -8 and even angiographic regression of the stented lesions. 8,9 Recently, Choussat et al 10 reported clinical stability of the stented target site at 8 to 10 years after coronary stenting. However, there is still no long-term angiographic data supporting sustained patency of the stented target site and absence of local complications specific for permanent implantation of metallic prosthesis.…”
mentioning
confidence: 99%
“…Choussat et al [1] reported that clinical stability of the stented target site is at 8-10 years after coronary stenting. By contrast, Kimura et al [2] reported that 7-11 years of angiographic followup demonstrated late luminal renarrowing beyond 4 years, which did not necessarily require target lesion PCI.…”
Section: Clinical Course Of Isr After Bare-metal Stent (Bms) Implantamentioning
confidence: 99%
“…Mechanistically, IL-6 contributes to CAD development by affecting metabolic, endothelial and coagulant events, and is viewed as a local and circulating marker of coronary plaque inflammation. IL-6 was implicated in the pathogenesis of ischemic cardiovascular events, including unstable angina11 and ACS,12 and its expression and secretion are regulated by IL-1 and TNF-α, which are highly induced in the atherosclerotic plaque 1314. IL-6 induces the expression of tissue factor, monocyte chemotactic protein-1, matrix-degrading enzyme and low-density lipoprotein receptors in macrophages, and stimulates the aggregation of platelets, proliferation of vascular smooth muscle cells and production of C-reactive protein and fibrinogen 1315…”
mentioning
confidence: 99%