&lt;b&gt;A Case Report of Very Late Thrombosis of Coronary Bare-Metal Stent 10 Years After Stenting&lt;/b&gt;

Katayama, Toshiro; Yamamoto, Tadashi; Iwasaki, Yoshihiro

doi:10.1536/ihj.48.547

Cited by 3 publications

(1 citation statement)

References 22 publications

Supporting

Mentioning

Contrasting

Order By: Relevance

“…However, a previous study has shown that >30% of patients with BMS in-stent restenosis require hospitalization due to the acute coronary syndrome (ACS) [2]. In addition, the development of atherosclerosis within the neointima has also been a cause of concern [3,4]. …”

Section: Introductionmentioning

confidence: 99%

Renal Dysfunction and Atherosclerosis of the Neointima following Bare Metal Stent Implantation

Hayano

Ishii²,

Ichimiya³

et al. 2013

Am J Nephrol

View full text Add to dashboard Cite

Background: Recently, neoatherosclerosis within the neointima after bare metal stent (BMS) implantation, which could cause late restenosis and very late stent thrombosis, has been a cause of concern. Renal dysfunction has been related to late cardiovascular events after coronary intervention. The present study was conducted focusing on the relationship between renal dysfunction and neointimal tissue characteristics with BMS restenosis using integrated backscatter intravascular ultrasound (IB-IVUS). Methods: We prospectively performed IB-IVUS in 80 consecutive patients requiring target lesion revascularization after BMS implantation; the patients were divided into two groups according to the estimated glomerular filtration [eGFR: ≥60 (n = 49) and <60 ml/min/1.73 m² (n = 31)]. Results: Patients with eGFR <60 ml/min/1.73 m² had a significantly higher percentage of lipid tissue volume within the neointima and a lower percentage of fibrous tissue volume than those with eGFR ≥60 ml/min/1.73 m² (23.2 ± 9.4 vs. 18.0 ± 7.0%, p = 0.005, and 75.3 ± 9.3 vs. 80.4 ± 7.0%, p = 0.007, respectively). Using logistic regression analysis, eGFR <60 ml/min/1.73 m² and duration from stent implantation ≥48 months were independent predictors of increased lipid tissue volume within the neointima (odds ratio, 3.93; 95% confidence interval, 1.15-13.46, p = 0.03, and odds ratio, 7.56; 95% confidence interval, 2.02-28.30, p = 0.003, respectively). Conclusions: Lower eGFR levels were associated with greater lipid tissue volume within the neointima after BMS deployment, suggesting the development of atherosclerosis. Renal dysfunction may affect neointimal tissue characteristics and thus leading to an increased risk of late stent failure.

show abstract

Section: Introductionmentioning

confidence: 99%

Renal Dysfunction and Atherosclerosis of the Neointima following Bare Metal Stent Implantation

Hayano

Ishii²,

Ichimiya³

et al. 2013

Am J Nephrol

View full text Add to dashboard Cite

show abstract

Very late thrombosis in a bare metal stent

Qian

Yang

2011

Cathet Cardio Intervent

View full text Add to dashboard Cite

Recent publications have emphasized the concept that restenosis of a bare metal stent (BMS) is not as clinically benign as previously believed. We present a case of very late stent thrombosis 9 years after implantation of a BMS is described. Optical coherence tomography shows plaque rupture in the stent. These findings suggest that the formation and progression of a neoatheroma over a previously healed intrastent vascular intima may represent a "new" mechanism for stent thrombosis. Evidence from the current case supports this hypothesis.

show abstract

Bare-metal stent thrombosis two decades after stenting

Acibuca

Gerede

Vurgun

2015

CVJA

View full text Add to dashboard Cite

Very late bare-metal stent (BMS) thrombosis is unusual in clinical practice. To the best of our knowledge, the latest that the thrombosis of a BMS has been reported is 14 years after implantation. Here, we describe a case of BMS thrombosis that occurred two decades after stenting. A 68-year-old male patient was admitted with acute anterior myocardial infarction. This patient had a history of BMS implantation in the left anterior descending coronary artery (LAD) 20 years previously. Immediate coronary angiography demonstrated acute thrombotic occlusion of the stent in the LAD. With this case, we are recording the latest reported incidence of BMS thrombosis after implantation.

show abstract

<b>A Case Report of Very Late Thrombosis of Coronary Bare-Metal Stent 10 Years After Stenting</b>

Cited by 3 publications

References 22 publications

Renal Dysfunction and Atherosclerosis of the Neointima following Bare Metal Stent Implantation

Renal Dysfunction and Atherosclerosis of the Neointima following Bare Metal Stent Implantation

Very late thrombosis in a bare metal stent

Bare-metal stent thrombosis two decades after stenting

Contact Info

Product

Resources

About