“…4 Fewer than 1% of AN bones are symptomatic, due to pressure of the bony prominence, bursitis, continuous irritation at the interface between the AN and the navicular, a degenerative or traumatic tear at the synchondrosis, flatfoot due to an abnormal tibialis posterior tendon (TPT) insertion, posterior tibial tendon dysfunction, fracture, osteitis, cartilage cap destruction, or osteonecrosis. 1,3,[5][6][7][8][9][10] Most of the symptomatic cases are type II AN bones and respond to conservative treatment. If conservative measures fail, surgery is indicated.…”