“…Deltoid ruptures have been reported in the setting of chronic RCTs, steroid injections, sclerotic changes, open RCR or acromioplasty, trauma, calcific tendinopathy, and chronic subacromial bursitis [ 3 – 6 ]. Sclerotic changes of the humeral greater tuberosity can accelerate the rate at which attrition occurs by creating frictional forces on the undersurface of the deltoid [ 3 ]. Calcific tendinopathy and chronic subacromial subdeltoid bursitis are two additional conditions where degeneration and chronic local inflammation, respectively, can lead to deltoid rupture [ 3 ].…”