1977
DOI: 10.1007/bf01683065
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Massive pulmonary embolism without arterial hypoxaemia

Abstract: Two cases of massive pulmonary embolism, confirmed by angiographic or necropsy findings, were remarkable by the absence of arterial hypoxaemia. The various mechanisms responsible for arterial hypoxaemia in pulmonary embolism are discussed. It is suggested that in patients with massive pulmonary embolism a markedly decreased cardiac output might account for the absence of arterial hypoxaemia. In the light of these two cases the finding of a normal PaO2 does not rule out the diagnosis of pulmonary embolism.

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Cited by 9 publications
(3 citation statements)
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“…In obstructive shock recalcitrant hypotension is not surprising; in fact, it's expected. 5 Tension pneumothorax is a classic cause of this kind of problem, which is usually seen in the context of penetrating trauma. Our patient's normal chest imaging studies completely eliminate this possibility.…”
Section: Faculty Discussionmentioning
confidence: 99%
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“…In obstructive shock recalcitrant hypotension is not surprising; in fact, it's expected. 5 Tension pneumothorax is a classic cause of this kind of problem, which is usually seen in the context of penetrating trauma. Our patient's normal chest imaging studies completely eliminate this possibility.…”
Section: Faculty Discussionmentioning
confidence: 99%
“…There are, though, a number of reports of unexpectedly high pO 2 in patients with massive PE and shock. 5,6 This may be due to low cardiac output with increased dwell time of blood in the pulmonic vasculature. Of note, stroke in association with PE is also reported.…”
Section: Faculty Discussionmentioning
confidence: 99%
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