Mean Target Intraocular Pressure and Progression Rates in Chronic Angle-Closure Glaucoma

Liza-Sharmini, Ahmad Tajudin; Yin, Nancy Yuen Shi; Lee, Shami Shi-Huang; Jackson, Angi L.; Stewart, William C.

doi:10.1089/jop.2008.0061

Cited by 15 publications

(15 citation statements)

References 19 publications

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“…This is similar to CACG patients' natural progression rate (25%), which regards mean target IOP as an important risk factor for progression. 20 However, 25% is a greater rate than 14.8%, which Tham et al 8 reported for medically uncontrolled CACG patients who had received a second glaucoma operation for progression. The reason for our greater percentage may result from our longer follow-up time and more strict exclusions in our retrospective study.…”

Section: Discussionmentioning

confidence: 94%

Risk Factors of Disease Progression After Cataract Surgery in Chronic Angle-closure Glaucoma Patients

Lee

Hong

et al. 2016

Journal of Glaucoma

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Section: Discussionmentioning

confidence: 94%

Risk Factors of Disease Progression After Cataract Surgery in Chronic Angle-closure Glaucoma Patients

Lee

Hong

et al. 2016

Journal of Glaucoma

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“…Recently, several studies have indicated that target pressure of low teens may be required for eyes with advanced PACG to prevent glaucoma progression. 23,24 It is likely that phacotrabeculectomy is a better option than PHCE alone for patients with advanced PACG who have a preoperative IOPXACD product greater than 35 while on multiple medications.…”

Section: Discussionmentioning

confidence: 99%

Determinants of Long-term Intraocular Pressure After Phacoemulsification in Primary Angle-closure Glaucoma

Liu

Cheng

et al. 2011

Journal of Glaucoma

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“…It is the abrupt increase in IOP that has been recognized as a critical stress factor for RGCs and their axons23141516.…”

mentioning

confidence: 99%

A rat experimental model of glaucoma incorporating rapid-onset elevation of intraocular pressure

Smędowski

Pietrucha‐Dutczak

Kaarniranta

et al. 2014

Sci Rep

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Glaucoma is a chronic disease that causes structural and functional damage to retinal ganglion cells (RGC). The currently employed therapeutic options are not sufficient to prevent vision loss in patients with glaucoma; therefore, there is a need to develop novel therapies, which requires the creation of functional, repeatable and easy-to-utilize animal models for use in pre-clinical studies. The currently available models ensure only low to moderate damage in optic nerves, with high variation in the outcomes and poor repeatability. We have developed an effective and reproducible rat glaucoma model based on a previous idea for a “Bead Model” in mice, which could be useful in future glaucoma research. Additionally, in an attempt to achieve rapid elevation of Intraocular Pressure (IOP), we included an initial “high-pressure injury” as part of this method, which serves as the equivalent of a severe glaucoma attack. These modifications made it possible to achieve longer lasting IOP elevation with chronic damage of retinal ganglion cells.

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Mean Target Intraocular Pressure and Progression Rates in Chronic Angle-Closure Glaucoma

Abstract: IOP reduction in CACG patients may help prevent progressive visual-field and optic-disc damage.

Cited by 15 publications

References 19 publications

Risk Factors of Disease Progression After Cataract Surgery in Chronic Angle-closure Glaucoma Patients

Risk Factors of Disease Progression After Cataract Surgery in Chronic Angle-closure Glaucoma Patients

Determinants of Long-term Intraocular Pressure After Phacoemulsification in Primary Angle-closure Glaucoma

A rat experimental model of glaucoma incorporating rapid-onset elevation of intraocular pressure

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