Measurement of foot artery blood pressure by micromanometry in normal subjects and in patients with arterial occlusive disease.

Bollinger, A; Barras, Jean‐Luc; Mahler, F

doi:10.1161/01.cir.53.3.506

Cited by 44 publications

(9 citation statements)

References 19 publications

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“…The main complex of vessels supplying the lower limb (common iliac-external iliac-femoral-popliteal-posterior tibial, Panels 18-22) features a triphasic flow contour which is a classical signature of the blood flow at those locations. The pressure pulse predicted at the posterior tibial artery (Panel 22) is very close to measurements published in [52].…”

Section: A Pressure and Flow Rate Waveformssupporting

confidence: 77%

An Anatomically Detailed Arterial Network Model for One-Dimensional Computational Hemodynamics

Blanco

Watanabe

Passos

et al. 2015

IEEE Trans. Biomed. Eng.

132

170

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Simulation platforms are increasingly becoming complementary tools for cutting-edge cardiovascular research. The interplay among structural properties of the arterial wall, morphometry, anatomy, wave propagation phenomena, and ultimately, cardiovascular diseases continues to be poorly understood. Accurate models are powerful tools to shed light on these open problems. We developed an anatomically detailed computational model of the arterial vasculature to conduct 1-D blood flow simulations to serve as simulation infrastructure to aid cardiovascular research. An average arterial vasculature of a man was outlined in 3-D space to serve as geometrical substrate for the mathematical model. The architecture of this model comprises almost every arterial vessel acknowledged in the medical/anatomical literature, with a resolution down to the luminal area of perforator arteries. Over 2000 arterial vessels compose the model. Anatomical, physiological, and mechanical considerations were employed for the set up of model parameters and to determine criteria for blood flow distribution. Computational fluid dynamics was used to simulate blood flow and wave propagation phenomena in such arterial network. A sensitivity analysis was developed to unveil the contributions of model parameters to the conformation of the pressure waveforms. In addition, parameters were modified to target model to a patient-specific scenario. On the light of the knowledge domain, we conclude that the present model features excellent descriptive and predictive capabilities in both patient-generic and patient-specific cases, presenting a new step toward integrating an unprecedented anatomical description, morphometric, and simulations data to help in understanding complex arterial blood flow phenomena and related cardiovascular diseases.

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Section: A Pressure and Flow Rate Waveformssupporting

confidence: 77%

An Anatomically Detailed Arterial Network Model for One-Dimensional Computational Hemodynamics

Blanco

Watanabe

Passos

et al. 2015

IEEE Trans. Biomed. Eng.

132

170

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“…10 An A/A index of less than 95% was found in 12 out of 99 persons (12%). No significant gender difference was found.…”

Section: Resultsmentioning

confidence: 95%

Asymptomatic carotid artery bruits in a population of elderly adults with isolated systolic hypertension.

Sutton¹,

Dai²,

Kuller³

1985

Stroke

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SUMMARY A sample of 99 elderly (over age sixty) adults with isolated systolic hypertension were examined for cervical arterial bruits and systolic blood pressure measurements were taken in upper and lower extremities. The ratio of ankle to arm blood pressure (ankle/arm index) was used as a measure of the degree of obstruction in peripheral arterial circulation. The prevalence of bruit (asymptomatic in all cases) was high, with 24 bruits detected in 19 (19%) participants. This high prevalence of bruits is thought to be due to the blood pressure characteristics of this population. Those with bruit were found to have a 3.9 times greater chance of having an abnormal ankle/arm index. The relationship remained statistically significant even after controlling for the effects of age and smoking. These findings add further evidence to the hypothesis that asymptomatic cervical bruits are an indication of systemic vascular disease. Stroke Vol 16, No 5, 1985THE RISK OF STROKE for persons with asymptomatic carotid bruit continues to be a very controversial topic. Two recent prospective studies have provided data suggesting that asymptomatic carotid bruits increase the risk of stroke between 1.6 and 7.5 fold.1 2However, correlation between location of the carotid bruit and type of subsequent stroke was poor. Myocardial infarction also occurred more frequently and to a degree similar to that observed for stroke. In general, mortality, particularly cardiovascular mortality, was notably higher in persons with asymptomatic carotid bruit. The frequency of carotid bruits has been found to be more than twice as high in persons with systolic hypertension as in normals.2 High systolic blood pressure has also been associated with other forms of vascular disease, including peripheral arterial stenosis.3 Little information however has been reported about the relationship between carotid bruits and peripheral arterial stenosis. The current research was designed to investigate this relationship in a group of 1.09 elderly (over age sixty) men and women with isolated systolic hypertension. MethodsThe subjects involved in the current investigation were participants enrolled in the Systolic Hypertension in the Elderly Program (SHEP), a multi-center clinical trial designed to test the efficacy of treatment of systolic hypertension in an elderly population. Screening for this study took place at shopping malls, retirement centers, churches, senior citizens' clubs and other locations where predominantly healthy elderly adults could be found. Qualifications for entry into the study included an age of 60 or above, systolic pressure greater than or equal to 160 and diastolic blood pressure less than 90. Exclusions for entry were as follows:1) Current treatment with antihypertensive medications.

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“…using plethysmography have shown flow diversion from the foot to the calf.l9 We suggest that the steep decrease of tCP02 (37 ° C) in some patients during intraarterial PGE, infusion is due to its strong vasodilatory potency with a similar blood flow diversion observed during postocclusive reactive hyperemia or intraarterial application of a mixture of energy-rich phosphates. 19,20 We were able to show that in most cases these alterations are persistent for at least two hours after the end of PGE, application, whereas in general after six hours the initial values had reestablished.…”

Section: Discussionmentioning

confidence: 77%

Skin Oxygen Pressure Histograms in Patients with Peripheral Arterial Occlusive Disease During Intraarterial and Intravenous Prostaglandin E1 Infusions of Different Dosages and Their Prognostic Value

et al. 1995

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Skin surface oxygen pressure fields (tcPO2 [37 degrees C]) reproducibly characterize skin microcirculation in patients with peripheral arterial occlusive disease. These appear suited for investigation of short- and long-term effects of vasoactive drug treatment. The authors studied whether skin surface oxygen pressure histograms change depending on dosage and route of administration of prostaglandin E1 (PGE1), whether they are of predictive value for patients' clinical outcome, and whether they normalize after therapy with PGE1. The authors investigated 15 patients with various degrees of disease and measured forefoot oxygen histograms consisting of at least 80 single tcPO2 (37 degrees C) values before and during intraarterial infusion (1.5, 3, or 6 ng/kg/minute) and intravenous infusion (4.5, or 9 ng/kg/minute). The measurements were repeated two and six hours after the end of intraarterial application of 1.5 ng/kg/minute. Furthermore, the orthostatic vasoconstrictor response was tested. Skin oxygen pressure histograms were controlled after a period of twenty-two (mean) days of intraarterial PGE1 therapy. Resting histograms were left shifted with median tcPO2 (37 degrees C) between 1 and 7 mm Hg. During intraarterial application, histograms were shifted to lower tcPO2 (37 degrees C) values in most patients. Only in 3 diabetic subjects with proximal or acral obliterations was a marked increase observed. The alterations were detectable at least two hours after the end of the infusion. During intravenous infusion, histograms did not change in most cases. After long-term therapy, histograms were substantially unchanged. A pathologic vasoconstrictor response, which was present in 10 patients, could not be restored. Despite a marked deterioration of the histograms the clinical outcome was favorable in 7 patients. Patients with a high resting tcPO2 (37 degrees C) (median 4 mm Hg and more) and those with a vasoconstriction on orthostasis are likely to respond to PGE1 therapy.

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Measurement of foot artery blood pressure by micromanometry in normal subjects and in patients with arterial occlusive disease.

Cited by 44 publications

References 19 publications

An Anatomically Detailed Arterial Network Model for One-Dimensional Computational Hemodynamics

An Anatomically Detailed Arterial Network Model for One-Dimensional Computational Hemodynamics

Asymptomatic carotid artery bruits in a population of elderly adults with isolated systolic hypertension.

Skin Oxygen Pressure Histograms in Patients with Peripheral Arterial Occlusive Disease During Intraarterial and Intravenous Prostaglandin E1 Infusions of Different Dosages and Their Prognostic Value

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