Meningiomas and Aneurysms of the Cavernous Sinus

Trobe, Jonathan D.; Glaser, Joel S.; Post, Judith D.

doi:10.1001/archopht.1978.03910050233009

Cited by 106 publications

(27 citation statements)

References 33 publications

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“…Previous cases of isolated inferior or superior division palsy of the oculomotor nerve due to trauma [10], tumors [11], and postinfectious neuropathy [9] have im plied that the site of damage is localized to the anterior portion of the cavernous sinus or in the orbit where the distal end of the nerve divides into the superior and infe rior branches [12], However, 5 reported cases of isolated superior division oculomotor nerve palsy caused by dis ease in the subarachnoid space suggested that the oculo motor nerve may maintain a divisional organization in its proximal course [13].…”

Section: Discussionmentioning

confidence: 99%

Inferior Division Paresis of the Oculomotor Nerve Caused by Basilar Artery Aneurysm

Kardon

Traynelis²,

Biller

1991

Cerebrovasc Dis

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A 60-year-old woman with progressive ataxia and progressive diplopia on downgaze was found to have a paresis of the inferior division oculomotor nerve. There was no ptosis or signs of aberrant regeneration. A large aneurysm at the tip of the basilar artery was discovered which invaginated the midbrain. Although inferior division oculomotor nerve palsy is rare and has not been considered to be caused by aneurysms, it should not always be considered benign, especially if there is progression or association with other neurologic signs. This case also helps to further delineate the topical Organization of the oculomotor nerve fascicle.

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Section: Discussionmentioning

confidence: 99%

Inferior Division Paresis of the Oculomotor Nerve Caused by Basilar Artery Aneurysm

Kardon

Traynelis²,

Biller

1991

Cerebrovasc Dis

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“…They differ from other intracranial aneurysms in that they are separated from the subarachnoid space by the dura of the cavernous sinus [17]. Consequently, they cause up to 25% of parasellar syndromes [11,18]. Patients may present with abrupt onset of facial pain or dysesthesia, abduction palsies, and oculosympathetic involvement, although any combination of ocular motility disruption can be seen [13,[17][18][19].…”

Section: Vascular Lesionsmentioning

confidence: 99%

“…Consequently, they cause up to 25% of parasellar syndromes [11,18]. Patients may present with abrupt onset of facial pain or dysesthesia, abduction palsies, and oculosympathetic involvement, although any combination of ocular motility disruption can be seen [13,[17][18][19]. Larger ICAs appear more likely to cause cranial nerve palsies [17,19].…”

Section: Vascular Lesionsmentioning

confidence: 99%

Parasellar syndromes

Johnston¹

2002

Curr Neurol Neurosci Rep

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The parasellar compartments are located lateral to and on either side of the sella turcica. The cavernous sinuses are the most prominent anatomic feature of the parasella. Each sinus consists of a plexus of veins through which runs the intracavernous portion of the internal carotid artery. Ocular motor nerves three and four travel within the dural covering of the cavernous sinus to the superior orbital fissure, and cranial nerve six travels through the carotid sinus itself, giving rise to parasellar syndromes, which have distinctive clinical features. Ophthalmoplegia occurs as a result of damage to these ocular motor nerves and variable involvement of oculosympathetic nerves. Facial pain, dysesthesia, and paraesthesia are caused by damage to one or more of the divisions of the fifth cranial nerve, travelling in the dural wall of the cavernous sinus. Tumors, such as meningiomas, frequently cause parasellar syndromes, as do aneurysms of the intracavernous portion of the internal carotid artery, carotid-cavernous fistulas, and cavernous sinus thrombosis. Inflammatory conditions such as Tolosa-Hunt syndrome, ischemia to small vessels supplying the cavernous portion of the cranial nerves, and infections can cause this syndrome. Magnetic resonance imaging is the investigation of choice and therapy is specific to the cause of the parasellar syndrome, but now includes more aggressive endoscopic and microsurgical intervention, and radiosurgery.

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“…Parasympathetic and sympathetic disturbances are present because of oculomotor nerve involvement and involvement of the sympathetic parasellar connections. However, the autonomic nerve disturbances may be obscured because of the simultaneous occurrence of parasympathetic and sympathetic disturbances (Trobe 1978). The conjunctival injection as part of a sympathetic involvement is hidden by the exophthalmus and venous congestion.…”

Section: The Superior Orbital Fissure Syndromementioning

confidence: 99%

The Cavernous Sinus Syndrome: An Anatomical and Clinical Study

Overbeeke¹

1991

Surgery of the Sellar Region and Paranasal Sinuses

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Meningiomas and Aneurysms of the Cavernous Sinus

Cited by 106 publications

References 33 publications

Inferior Division Paresis of the Oculomotor Nerve Caused by Basilar Artery Aneurysm

Inferior Division Paresis of the Oculomotor Nerve Caused by Basilar Artery Aneurysm

Parasellar syndromes

The Cavernous Sinus Syndrome: An Anatomical and Clinical Study

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