Metastatic choroidal abscess and choroidal neovascularization in a patient with Staphylococcus aureus renal abscess

Cw, Wu; Cs, Yang; Chang, Hyo Won; Fl, Lee; Wm, Hsu

doi:10.1038/sj.eye.6701843

Cited by 6 publications

(2 citation statements)

References 9 publications

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“…Most of the cases reported were caused by endogenous infections by bacteria, predominantly Klebsiella pneumoniae [1] and Nocardia spp. [2], and occasionally Pseudomonas aeruginosa [3], Streptococcus aureus [4], and Streptococcus viridans [5]. In comparison, a fungal subretinal abscess is a considerably more uncommon condition, with only six cases caused by Aspergillus spp.…”

Section: Introductionmentioning

confidence: 95%

Endogenous Candida albicans infection causing subretinal abscess

et al. 2009

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Section: Introductionmentioning

confidence: 95%

Endogenous Candida albicans infection causing subretinal abscess

et al. 2009

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“…CNV can be a rare sequela of bacterial infectious choroiditis. [23][24][25][26][27][28][29][30][31][32] Choroidal colonisation by the bacteria can occur after bacterial metastasis, such as endocarditis, aortic valve infection, renal and bone abscesses and intravenous drug abuse. CNVs secondary to bacterial choroiditis are generally membranes that grow close to the primary chorioretinal lesion or in the neighbouring area of an old atrophic scar.…”

mentioning

confidence: 99%

New Trends in the Management of Inflammatory Choroidal Neovascularisation

Neri¹

2009

European Ophthalmic Review

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Choroidal neovascularisation (CNV) can be a severe, sight-threatening sequela in patients with uveitis. CNV can occur in both infectious and non-infectious diseases. In the majority of cases, fluorescein angiography, indocyanine green angiography and optical coherence tomography allow the clinical characteristics of the CNV to be accurately determined. An infectious disease should be looked for so that patients can be given a suitable therapy when available. Regarding non-infectious inflammatory CNV, the treatment strategy should be aimed at controlling inflammation. Systemic corticosteroids with or without immunosuppressants are suggested, even when the CNV occurs with apparently inactive uveitis. In this case, chronic sub-clinical inflammation can be the basis for the CNV pathogenesis. Additional therapies aimed directly at the neovascular process, such as intravitreal antivascular endothelial growth factor agents, are recommended, particularly when the therapy shows insufficient efficacy. However, the current data are still only based on case reports or small series. For such reasons, further trials are mandatory to validate the preliminary results.

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